Samuel E. Gandy, MD PhD
Affiliations: | Neurology and Psychiatry | Icahn School of Medicine at Mount Sinai, New York, NY, United States |
Area:
http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=74Website:
http://www.mountsinai.org/profiles/samuel-e-gandy?id=0000072500003639565131Google:
"Samuel Gandy"Cross-listing: Neurotree
Children
Sign in to add trainee| John W. Steele | grad student | 2007-2011 | Mount Sinai (Neurotree) |
| Hannah Brautigam | grad student | 2013 | Mount Sinai School of Medicine (Neurotree) |
| Odete A. Beirão da Cruz e Silva | post-doc | Rockefeller |
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Publications
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| Beckmann ND, Lin WJ, Wang M, et al. (2020) Multiscale causal networks identify VGF as a key regulator of Alzheimer's disease. Nature Communications. 11: 3942 |
| Kritikos M, Clouston SAP, Diminich ED, et al. (2020) Pathway Analysis for Plasma β-Amyloid, Tau and Neurofilament Light (ATN) in World Trade Center Responders at Midlife. Neurology and Therapy |
| Woo YJ, Roussos P, Haroutunian V, et al. (2020) Comparison of brain connectomes by MRI and genomics and its implication in Alzheimer's disease. Bmc Medicine. 18: 23 |
| El Gaamouch F, Audrain M, Lin WJ, et al. (2020) VGF-derived peptide TLQP-21 modulates microglial function through C3aR1 signaling pathways and reduces neuropathology in 5xFAD mice. Molecular Neurodegeneration. 15: 4 |
| Readhead B, Haure-Mirande JV, Ehrlich ME, et al. (2019) Clarifying the Potential Role of Microbes in Alzheimer's Disease. Neuron. 104: 1036-1037 |
| Hata S, Omori C, Kimura A, et al. (2019) Decrease in p3-Alcβ37 and p3-Alcβ40, products of Alcadein β generated by γ-secretase cleavages, in aged monkeys and patients with Alzheimer's disease. Alzheimer's & Dementia (New York, N. Y.). 5: 740-750 |
| Clouston SAP, Deri Y, Diminich E, et al. (2019) Posttraumatic stress disorder and total amyloid burden and amyloid-β 42/40 ratios in plasma: Results from a pilot study of World Trade Center responders. Alzheimer's & Dementia (Amsterdam, Netherlands). 11: 216-220 |
| Haure-Mirande JV, Wang M, Audrain M, et al. (2018) Correction: Integrative approach to sporadic Alzheimer's disease: deficiency of TYROBP in cerebral Aβ amyloidosis mouse normalizes clinical phenotype and complement subnetwork molecular pathology without reducing Aβ burden. Molecular Psychiatry |
| Haure-Mirande JV, Wang M, Audrain M, et al. (2018) Integrative approach to sporadic Alzheimer's disease: deficiency of TYROBP in cerebral Aβ amyloidosis mouse normalizes clinical phenotype and complement subnetwork molecular pathology without reducing Aβ burden. Molecular Psychiatry |
| Audrain M, Haure-Mirande JV, Wang M, et al. (2018) Integrative approach to sporadic Alzheimer's disease: deficiency of TYROBP in a tauopathy mouse model reduces C1q and normalizes clinical phenotype while increasing spread and state of phosphorylation of tau. Molecular Psychiatry |