Robert A. Cornell
Affiliations: | Anatomy & Cell Biology | University of Iowa, Iowa City, IA |
Area:
Human Development, Genetics, Neuroscience BiologyGoogle:
"Robert Cornell"Children
Sign in to add trainee| Wei Li | grad student | 2002-2008 | University of Iowa Carver College of Medicine (FlyTree) |
| Matthew S. McNeill | grad student | 2009 | University of Iowa |
| Eric S. Van Otterloo | grad student | 2012 | University of Iowa |
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Publications
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| Nguyen TT, Mitchell JM, Kiel MD, et al. (2023) TFAP2 paralogs regulate midfacial development in part through a conserved ALX genetic pathway. Development (Cambridge, England) |
| Kenny C, Dilshat R, Seberg HE, et al. (2022) TFAP2 paralogs facilitate chromatin access for MITF at pigmentation and cell proliferation genes. Plos Genetics. 18: e1010207 |
| Kumari P, Sturgeon M, Bonde G, et al. (2022) Generating Zebrafish RNA-Less Mutant Alleles by Deleting Gene Promoters with CRISPR/Cas9. Methods in Molecular Biology (Clifton, N.J.). 2403: 91-106 |
| Hamm M, Sohier P, Petit V, et al. (2021) BRN2 is a non-canonical melanoma tumor-suppressor. Nature Communications. 12: 3707 |
| Dilshat R, Fock V, Kenny C, et al. (2021) MITF reprograms the extracellular matrix and focal adhesion in melanoma. Elife. 10 |
| He M, Zuo X, Liu H, et al. (2020) Genome-wide Analyses Identify a Novel Risk Locus for Nonsyndromic Cleft Palate. Journal of Dental Research. 22034520943867 |
| Zhang M, Zhang J, Zhao H, et al. (2020) Functional Characterization of a Novel IRF6 Frameshift Mutation From a Van Der Woude Syndrome Family. Frontiers in Genetics. 11: 562 |
| Liu H, Duncan K, Helverson A, et al. (2020) Analysis of zebrafish periderm enhancers facilitates identification of a regulatory variant near human . Elife. 9 |
| Liu H, Duncan K, Helverson A, et al. (2020) Author response: Analysis of zebrafish periderm enhancers facilitates identification of a regulatory variant near human KRT8/18 Elife |
| Cox TC, Lidral AC, McCoy JC, et al. (2019) Mutations in GDF11 and the extracellular antagonist, Follistatin, as a likely cause of Mendelian forms of orofacial clefting in humans. Human Mutation |