Kali Janes, Ph.D.
Affiliations: | 2013 | Pharmacological and Physiological Science | Saint Louis University, St. Louis, MO, United States |
Area:
Pharmacology, Physiology Biology, Neuroscience BiologyGoogle:
"Kali Janes"Parents
Sign in to add mentor| Daniela Salvemini | grad student | 2013 | Saint Louis University | |
| (A3 adenosine receptor agonist as novel therapeutic agents to prevent and reverse chemotherapy-induced neuropathic pain.) | ||||
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| Doyle TM, Hutchinson MR, Braden K, et al. (2020) Sphingosine-1-phosphate receptor subtype 1 activation in the central nervous system contributes to morphine withdrawal in rodents. Journal of Neuroinflammation. 17: 314 |
| Doyle TM, Janes K, Chen Z, et al. (2020) Activation of sphingosine-1-phosphate receptor subtype 1 in the central nervous system contributes to morphine-induced hyperalgesia and antinociceptive tolerance in rodents. Pain |
| Stockstill K, Doyle TM, Yan X, et al. (2018) Dysregulation of sphingolipid metabolism contributes to bortezomib-induced neuropathic pain. The Journal of Experimental Medicine |
| Wahlman C, Doyle TM, Little JW, et al. (2018) Chemotherapy-induced pain is promoted by enhanced spinal adenosine kinase levels via astrocyte-dependent mechanisms. Pain |
| Janes K, Symons-Liguori AM, Jacobson KA, et al. (2016) Identification of A3 adenosine receptor agonists as novel non-narcotic analgesics. British Journal of Pharmacology |
| Little JW, Ford A, Symons-Liguori AM, et al. (2015) Endogenous adenosine A3 receptor activation selectively alleviates persistent pain states. Brain : a Journal of Neurology. 138: 28-35 |
| Janes K, Wahlman C, Little JW, et al. (2015) Spinal neuroimmune activation is independent of T-cell infiltration and attenuated by A3 adenosine receptor agonists in a model of oxaliplatin-induced peripheral neuropathy. Brain, Behavior, and Immunity. 44: 91-9 |
| Little J, Janes K, Chen Z, et al. (2015) (326) Supraspinal adenosine A3 receptor (A3AR) activation reverses chemotherapy-induced neuropathic pain through an IL-10 dependent mechanism The Journal of Pain. 16: S57 |
| Janes K, Esposito E, Doyle T, et al. (2014) A3 adenosine receptor agonist prevents the development of paclitaxel-induced neuropathic pain by modulating spinal glial-restricted redox-dependent signaling pathways. Pain. 155: 2560-7 |
| Janes K, Little JW, Li C, et al. (2014) The development and maintenance of paclitaxel-induced neuropathic pain require activation of the sphingosine 1-phosphate receptor subtype 1. The Journal of Biological Chemistry. 289: 21082-97 |