Anthony G. Passerini
Affiliations: | Biomedical Engineering | University of California, Davis, Davis, CA |
Area:
Biomedical Engineering, Biomechanics BiophysicsGoogle:
"Anthony Passerini"
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Publications
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| Li T, Conroy KL, Kim AM, et al. (2023) Role of MEF2C in the Endothelial Cells Derived from Human Induced Pluripotent Stem Cells. Stem Cells (Dayton, Ohio) |
| Fan X, Li Q, Wang Y, et al. (2022) Non-canonical NF-κB contributes to endothelial pyroptosis and atherogenesis dependent on IRF-1. Translational Research : the Journal of Laboratory and Clinical Medicine |
| Wang Y, Li Q, Zhang Z, et al. (2022) mTOR contributes to endothelium-dependent vasorelaxation by promoting eNOS expression and preventing eNOS uncoupling. Communications Biology. 5: 726 |
| Chen X, Hu C, Fan X, et al. (2021) mTOR Inhibition Promotes Pneumonitis Through Inducing Endothelial Contraction and Hyperpermeability. American Journal of Respiratory Cell and Molecular Biology |
| Hu C, Peng K, Wu Q, et al. (2021) HDAC1 and 2 regulate endothelial VCAM-1 expression and atherogenesis by suppressing methylation of the promoter. Theranostics. 11: 5605-5619 |
| Hernandez AA, Foster GA, Soderberg SR, et al. (2020) An Allosteric Shift in CD11c Affinity Activates a Proatherogenic State in Arrested Intermediate Monocytes. Journal of Immunology (Baltimore, Md. : 1950) |
| Peng K, Fan X, Li Q, et al. (2020) IRF-1 mediates the suppressive effects of mTOR inhibition on arterial endothelium. Journal of Molecular and Cellular Cardiology |
| Rand AA, Rajamani A, Kodani SD, et al. (2019) Epoxyeicosatrienoic acid (EET)-stimulated angiogenesis is mediated by epoxy hydroxyeicosatrienoic acids (EHETs) formed from COX-2. Journal of Lipid Research |
| Bailey KA, Moreno E, Haj FG, et al. (2019) Mechanoregulation of p38 activity enhances endoplasmic reticulum stress-mediated inflammation by arterial endothelium. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201900236R |
| Rajamani A, Borkowski K, Akre S, et al. (2019) Oxylipins in triglyceride-rich lipoproteins of dyslipidemic subjects promote endothelial inflammation following a high fat meal. Scientific Reports. 9: 8655 |