B Rita Alevriadou
Affiliations: | Biomedical Engineering | Ohio State University, Columbus, Columbus, OH |
Area:
Biomedical EngineeringGoogle:
"B Alevriadou"
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Publications
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| Patel A, Pietromicca JG, Venkatesan M, et al. (2023) Modulation of the mitochondrial Ca uniporter complex subunit expression by different shear stress patterns in vascular endothelial cells. Physiological Reports. 11: e15588 |
| Patel A, Simkulet M, Maity S, et al. (2022) The mitochondrial Ca uniporter channel synergizes with fluid shear stress to induce mitochondrial Ca oscillations. Scientific Reports. 12: 21161 |
| Alevriadou BR, Patel A, Noble M, et al. (2020) Molecular Nature and Physiological Role of the Mitochondrial Calcium Uniporter Channel. American Journal of Physiology. Cell Physiology |
| Moshkforoush A, Ashenagar B, Tsoukias NM, et al. (2019) Modeling the role of endoplasmic reticulum-mitochondria microdomains in calcium dynamics. Scientific Reports. 9: 17072 |
| Alevriadou BR, Shanmughapriya S, Patel A, et al. (2017) Mitochondrial Ca2+ transport in the endothelium: regulation by ions, redox signalling and mechanical forces. Journal of the Royal Society, Interface. 14 |
| Scheitlin CG, Julian JA, Shanmughapriya S, et al. (2016) Endothelial mitochondria regulate the intracellular Ca2+ response to fluid shear stress. American Journal of Physiology. Cell Physiology. ajpcell.00171.2015 |
| Scheitlin CG, Nair DM, Crestanello JA, et al. (2014) Fluid Mechanical Forces and Endothelial Mitochondria: A Bioengineering Perspective. Cellular and Molecular Bioengineering. 7: 483-496 |
| Giedt RJ, Pfeiffer DR, Matzavinos A, et al. (2012) Mitochondrial dynamics and motility inside living vascular endothelial cells: role of bioenergetics. Annals of Biomedical Engineering. 40: 1903-16 |
| Giedt RJ, Yang C, Zweier JL, et al. (2012) Mitochondrial fission in endothelial cells after simulated ischemia/reperfusion: role of nitric oxide and reactive oxygen species. Free Radical Biology & Medicine. 52: 348-56 |
| Chen J, Chen CL, Alevriadou BR, et al. (2011) Excess no predisposes mitochondrial succinate-cytochrome c reductase to produce hydroxyl radical. Biochimica Et Biophysica Acta. 1807: 491-502 |