Jonathan A. Kirk, Ph.D.

Affiliations: 
2009 University of Pittsburgh, Pittsburgh, PA, United States 
Area:
Biomedical Engineering
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"Jonathan Kirk"

Parents

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Sanjeev G. Shroff grad student 2009 University of Pittsburgh
 (Effects of protein kinase C phosphorylation of cardiac troponin I: An experimental and model-based study.)
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Publications

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Wang J, Tomar D, Martin TG, et al. (2023) Bag3 Regulates Mitochondrial Function and the Inflammasome Through Canonical and Noncanonical Pathways in the Heart. Jacc. Basic to Translational Science. 8: 820-839
Eaton DM, Martin TG, Kasa M, et al. (2022) HDAC Inhibition Regulates Cardiac Function by Increasing Myofilament Calcium Sensitivity and Decreasing Diastolic Tension. Pharmaceutics. 14
DiNello E, Bovo E, Thuo P, et al. (2020) Deletion of cardiac polycystin 2/PC2 results in increased SR calcium release and blunted adrenergic reserve. American Journal of Physiology. Heart and Circulatory Physiology
Martin TG, Kirk JA. (2020) Under construction: The dynamic assembly, maintenance, and degradation of the cardiac sarcomere. Journal of Molecular and Cellular Cardiology
Ai L, Perez E, Asimes A, et al. (2020) Binge Alcohol Exposure in Adolescence Impairs Normal Heart Growth. Journal of the American Heart Association. e015611
Pitoulis FG, Hasan W, Papadaki M, et al. (2020) Intact myocardial preparations reveal intrinsic transmural heterogeneity in cardiac mechanics. Journal of Molecular and Cellular Cardiology
Seflova J, Pribadi M, Kirk J, et al. (2020) Insight into Sodium Pump Regulation in the Failing Human Heart Biophysical Journal. 118: 129a
Stachowski MJ, Zlobin A, Papadaki M, et al. (2020) An Independent Pool of GSK-3β Modulates Calcium Sensitivity at the Cardiac Myofilament Biophysical Journal. 118: 592a-593a
Martin TG, Myers V, Willis M, et al. (2019) Abstract 909: Determining the Role of the Co-chaperone BAG3 at the Cardiac Myofilament Circulation Research. 125
Rattanasopa C, Kirk JA, Bupha-Intr T, et al. (2018) Estrogen but not testosterone preserves myofilament function from doxorubicin-induced cardiotoxicity by reducing oxidative modifications. American Journal of Physiology. Heart and Circulatory Physiology
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