Jonathan A. Kirk, Ph.D.
Affiliations: | 2009 | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
Biomedical EngineeringGoogle:
"Jonathan Kirk"Parents
Sign in to add mentorSanjeev G. Shroff | grad student | 2009 | University of Pittsburgh | |
(Effects of protein kinase C phosphorylation of cardiac troponin I: An experimental and model-based study.) |
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Publications
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Wang J, Tomar D, Martin TG, et al. (2023) Bag3 Regulates Mitochondrial Function and the Inflammasome Through Canonical and Noncanonical Pathways in the Heart. Jacc. Basic to Translational Science. 8: 820-839 |
Eaton DM, Martin TG, Kasa M, et al. (2022) HDAC Inhibition Regulates Cardiac Function by Increasing Myofilament Calcium Sensitivity and Decreasing Diastolic Tension. Pharmaceutics. 14 |
DiNello E, Bovo E, Thuo P, et al. (2020) Deletion of cardiac polycystin 2/PC2 results in increased SR calcium release and blunted adrenergic reserve. American Journal of Physiology. Heart and Circulatory Physiology |
Martin TG, Kirk JA. (2020) Under construction: The dynamic assembly, maintenance, and degradation of the cardiac sarcomere. Journal of Molecular and Cellular Cardiology |
Ai L, Perez E, Asimes A, et al. (2020) Binge Alcohol Exposure in Adolescence Impairs Normal Heart Growth. Journal of the American Heart Association. e015611 |
Pitoulis FG, Hasan W, Papadaki M, et al. (2020) Intact myocardial preparations reveal intrinsic transmural heterogeneity in cardiac mechanics. Journal of Molecular and Cellular Cardiology |
Seflova J, Pribadi M, Kirk J, et al. (2020) Insight into Sodium Pump Regulation in the Failing Human Heart Biophysical Journal. 118: 129a |
Stachowski MJ, Zlobin A, Papadaki M, et al. (2020) An Independent Pool of GSK-3β Modulates Calcium Sensitivity at the Cardiac Myofilament Biophysical Journal. 118: 592a-593a |
Martin TG, Myers V, Willis M, et al. (2019) Abstract 909: Determining the Role of the Co-chaperone BAG3 at the Cardiac Myofilament Circulation Research. 125 |
Rattanasopa C, Kirk JA, Bupha-Intr T, et al. (2018) Estrogen but not testosterone preserves myofilament function from doxorubicin-induced cardiotoxicity by reducing oxidative modifications. American Journal of Physiology. Heart and Circulatory Physiology |