Eun-Young Ahn, Ph.D.

Affiliations: 
2003 University of Alabama, Birmingham, Birmingham, AL, United States 
Area:
Pathology, Cell Biology, Molecular Biology
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"Eun-Young Ahn"

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Jay M. McDonald grad student 2003 UAB
 (Molecular mechanisms of apoptosis in human cholangiocarcinoma: Regulation by Fas, calcium /calmodulin and interferon -gamma.)
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Publications

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Murphy JM, Rodriguez YAR, Jeong K, et al. (2020) Targeting focal adhesion kinase in cancer cells and the tumor microenvironment. Experimental & Molecular Medicine
DeKelver RC, Yan M, Ahn EY, et al. (2013) Attenuation of AML1-ETO cellular dysregulation correlates with increased leukemogenic potential. Blood. 121: 3714-7
Lo MC, Peterson LF, Yan M, et al. (2012) Combined gene expression and DNA occupancy profiling identifies potential therapeutic targets of t(8;21) AML. Blood. 120: 1473-84
Ahn E, Higashi T, Yan M, et al. (2012) SON Regulates GATA-2 Through Transcriptional Control of the Mir-23a∼27a∼24-2 Cluster Blood. 120: 110-110
Matsuura S, Yan M, Ahn E, et al. (2010) Negative Effects of GM-CSF Signaling In t(8;21)-Induced Leukemia Blood. 116: 3163-3163
Pawar P, Ma L, Byon CH, et al. (2009) Molecular mechanisms of tamoxifen therapy for cholangiocarcinoma: role of calmodulin. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research. 15: 1288-96
Yan M, Ahn EY, Hiebert SW, et al. (2009) RUNX1/AML1 DNA-binding domain and ETO/MTG8 NHR2-dimerization domain are critical to AML1-ETO9a leukemogenesis. Blood. 113: 883-6
Ahn EY, Yan M, Malakhova OA, et al. (2008) Disruption of the NHR4 domain structure in AML1-ETO abrogates SON binding and promotes leukemogenesis. Proceedings of the National Academy of Sciences of the United States of America. 105: 17103-8
Park SE, Lee SW, Hossain MA, et al. (2008) A chenodeoxycholic derivative, HS-1200, induces apoptosis and cell cycle modulation via Egr-1 gene expression control on human hepatoma cells. Cancer Letters. 270: 77-86
Dayyani F, Wang J, Yeh JR, et al. (2008) Loss of TLE1 and TLE4 from the del(9q) commonly deleted region in AML cooperates with AML1-ETO to affect myeloid cell proliferation and survival. Blood. 111: 4338-47
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