Dan Feng, Ph.D.
Affiliations: | 2013 | Cell and Molecular Biology | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Molecular Biology, GeneticsGoogle:
"Dan Feng"Parents
Sign in to add mentorMitchell A. Lazar | grad student | 2013 | Penn | |
(Epigenomic and transcriptional regulation of hepatic metabolism by REV-ERB and HDAC3.) |
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Publications
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Zhang Y, Papazyan R, Damle M, et al. (2017) The hepatic circadian clock fine-tunes the lipogenic response to feeding through RORα/γ. Genes & Development |
Hong S, Zhou W, Fang B, et al. (2016) Dissociation of muscle insulin sensitivity from exercise endurance in mice by HDAC3 depletion. Nature Medicine |
Zhang Y, Fang B, Emmett MJ, et al. (2015) GENE REGULATION. Discrete functions of nuclear receptor Rev-erbα couple metabolism to the clock. Science (New York, N.Y.). 348: 1488-92 |
Fang B, Everett LJ, Jager J, et al. (2014) Circadian enhancers coordinate multiple phases of rhythmic gene transcription in vivo. Cell. 159: 1140-52 |
Lee JM, Wagner M, Xiao R, et al. (2014) Nutrient-sensing nuclear receptors coordinate autophagy. Nature. 516: 112-5 |
Sun Z, Feng D, Fang B, et al. (2013) Deacetylase-independent function of HDAC3 in transcription and metabolism requires nuclear receptor corepressor. Molecular Cell. 52: 769-82 |
Gerhart-Hines Z, Feng D, Emmett MJ, et al. (2013) The nuclear receptor Rev-erbα controls circadian thermogenic plasticity. Nature. 503: 410-3 |
Feng D, Lazar MA. (2012) Clocks, metabolism, and the epigenome. Molecular Cell. 47: 158-67 |
Bugge A, Feng D, Everett LJ, et al. (2012) Rev-erbα and Rev-erbβ coordinately protect the circadian clock and normal metabolic function. Genes & Development. 26: 657-67 |
Mullican SE, Gaddis CA, Alenghat T, et al. (2011) Histone deacetylase 3 is an epigenomic brake in macrophage alternative activation. Genes & Development. 25: 2480-8 |