Alina Molchadsky, Ph.D.
Affiliations: | 2011 | Weizmann Institute of Science, Rehovot, Israel |
Area:
Cell Biology, Biochemistry, Molecular BiologyGoogle:
"Alina Molchadsky"Parents
Sign in to add mentorVarda Rotter | grad student | 2011 | Weizmann Institute | |
(Characterization of the roles of p53 in mesenchymal cell differentiation; a focus on adipogenesis.) |
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Publications
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Koifman G, Shetzer Y, Eizenberger S, et al. (2018) A mutant p53-dependent embryonic stem cell gene signature is associated with augmented tumorigenesis of stem cells. Cancer Research |
Charni-Natan M, Solomon H, Molchadsky A, et al. (2018) Various stress stimuli rewire the profile of liver secretome in a p53-dependent manner. Cell Death & Disease. 9: 647 |
Solomon H, Dinowitz N, Pateras IS, et al. (2018) Mutant p53 gain of function underlies high expression levels of colorectal cancer stem cells markers. Oncogene |
Prokocimer M, Molchadsky A, Rotter V. (2017) Dysfunctional diversity of p53 proteins in adult acute myeloid leukemia: projections on diagnostic work-up and therapy. Blood |
Molchadsky A, Rotter V. (2017) p53 and its mutants on the slippery road from stemness to carcinogenesis. Carcinogenesis |
Grespi F, Landré V, Molchadsky A, et al. (2016) Differential regulated microRNA by wild type and mutant p53 in induced pluripotent stem cells. Cell Death & Disease. 7: e2567 |
Shetzer Y, Napchan Y, Kaufman T, et al. (2016) Immune deficiency augments the prevalence of p53 loss of heterozygosity in spontaneous tumors but not bi-directional loss of heterozygosity in bone marrow progenitors. International Journal of Cancer |
Charni M, Aloni-Grinstein R, Molchadsky A, et al. (2016) p53 on the crossroad between regeneration and cancer. Cell Death and Differentiation |
Shetzer Y, Molchadsky A, Rotter V. (2016) Oncogenic Mutant p53 Gain of Function Nourishes the Vicious Cycle of Tumor Development and Cancer Stem-Cell Formation. Cold Spring Harbor Perspectives in Medicine |
Charni M, Molchadsky A, Goldstein I, et al. (2015) Novel p53 target genes secreted by the liver are involved in non-cell-autonomous regulation. Cell Death and Differentiation |