Kirk M. Habegger, Ph.D.
Affiliations: | 2009 | Biochemistry and Molecular Biology | Indiana University, Bloomington, Bloomington, IN, United States |
Area:
Physiology Biology, Cell BiologyGoogle:
"Kirk Habegger"Parents
Sign in to add mentor| Jeffrey S. Elmendorf | grad student | 2009 | Indiana University | |
| (Membrane cholesterol balance in exercise and insulin resistance.) | ||||
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Publications
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| Covert JD, Grice BA, Thornburg MG, et al. (2023) An early, reversible cholesterolgenic etiology of diet-induced insulin resistance. Molecular Metabolism. 72: 101715 |
| Kim T, Nason S, Antipenko J, et al. (2022) Hepatic TORC2 Signaling Facilitates Acute Glucagon-Receptor Enhancement of Insulin-Stimulated Glucose Homeostasis in Mice. Diabetes |
| Habegger K. (2022) Crosstalk Between Insulin- and Glucagon-Receptor Signaling in the Hepatocyte. Diabetes |
| Sammy MJ, Connelly AW, Brown JA, et al. (2021) Mito-Mendelian Interactions Alter in vivo Glucose Metabolism and Insulin Sensitivity in Healthy Mice. American Journal of Physiology. Endocrinology and Metabolism |
| Zeigerer A, Sekar R, Kleinert M, et al. (2021) Glucagon's Metabolic Action in Health and Disease. Comprehensive Physiology. 11: 1759-1783 |
| Brahma MK, Ha CM, Pepin ME, et al. (2020) Increased Glucose Availability Attenuates Myocardial Ketone Body Utilization. Journal of the American Heart Association. 9: e013039 |
| Ren G, Kim T, Kim HS, et al. (2020) A Small Molecule, UAB126, Reverses Diet-Induced Obesity and its Associated Metabolic Disorders. Diabetes |
| Müller TD, Habegger K. (2020) Revisiting the Pharmacological Value of Glucagon: An Editorial for the Special Issue "The Biology and Pharmacology of Glucagon". International Journal of Molecular Sciences. 21 |
| Kim T, Nason SR, Antipenko J, et al. (2020) SAT-655 Bile Acid Sequestration Synergistically Accelerates Glucagon Receptor-Stimulated Body Weight Loss in Diet-Induced Obese Mice Journal of the Endocrine Society. 4 |
| Nason SR, Kim T, Antipenko JP, et al. (2019) Glucagon-Receptor Signaling Reverses Hepatic Steatosis Independent of Leptin Receptor Expression. Endocrinology |