Anthony Quinn
Affiliations: | Biology (Cell-Molecular Biology) | The University of Toledo |
Area:
Immunology, Molecular BiologyGoogle:
"Anthony Quinn"Children
Sign in to add trainee| Rhea Y. Busick | grad student | 2006 | The University of Toledo |
| Stella A. Mayo | grad student | 2006 | The University of Toledo |
| Sarah Rasche | grad student | 2010 | The University of Toledo |
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Publications
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| Nandedkar-Kulkarni N, Vartak AR, Sucheck SJ, et al. (2019) Development of a Bioconjugate Platform for Modifying the Immune Response of Autoreactive Cytotoxic T Lymphocytes Involved in Type 1 Diabetes. Bioconjugate Chemistry |
| Marrero I, Aguilera C, Hamm DE, et al. (2016) High-throughput sequencing reveals restricted TCR Vβ usage and public TCRβ clonotypes among pancreatic lymph node memory CD4(+) T cells and their involvement in autoimmune diabetes. Molecular Immunology. 74: 82-95 |
| Clark MP, Leaman DW, Hazelhurst LA, et al. (2015) An aza-anthrapyrazole negatively regulates Th1 activity and suppresses experimental autoimmune encephalomyelitis. International Immunopharmacology. 31: 74-87 |
| Kiraly A, Koffman B, Hacker M, et al. (2011) A novel aza-anthrapyrazole blocks the progression of experimental autoimmune encephalomyelitis after the priming of autoimmunity. Clinical Immunology (Orlando, Fla.). 141: 304-16 |
| Rasche SS, Phillips M, McInerney MF, et al. (2011) IL-13Rα1 expression on β-cell-specific T cells in NOD mice. Diabetes. 60: 1716-25 |
| Linkes S, Fry C, Quinn A. (2010) Antigen-Experienced CD4lo T Cells Are Linked to Deficient Contraction of the Immune Response in Autoimmune Diabetes. Autoimmune Diseases. 2010: 920148 |
| Quinn A. (2009) Are T-cell responses to GAD65 influential in type 1 diabetes? Diabetes. 58: 2729-30 |
| Rasche S, Busick RY, Quinn A. (2009) GAD65-Specific Cytotoxic T Lymphocytes Mediate Beta-Cell Death and Loss of Function. The Review of Diabetic Studies : Rds. 6: 43-53 |
| Dai YD, Jensen KP, Marrero I, et al. (2008) N-terminal flanking residues of a diabetes-associated GAD65 determinant are necessary for activation of antigen-specific T cells in diabetes-resistant mice. European Journal of Immunology. 38: 968-76 |
| Busick RY, Aguilera C, Quinn A. (2007) Dominant CTL-inducing epitopes on GAD65 are adjacent to or overlap with dominant Th-inducing epitopes. Clinical Immunology (Orlando, Fla.). 122: 298-311 |