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Lisa K. Busch, Ph.D.

Affiliations: 
2001 University of Iowa, Iowa City, IA 
Area:
Molecular Biology, Immunology
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"Lisa Busch"
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Gail A. Bishop grad student 2001 University of Iowa
 (Molecular mechanisms of LMP1 signaling to B lymphocytes.)

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Gumá M, Busch LK, Salazar-Fontana LI, et al. (2005) The CD94/NKG2C killer lectin-like receptor constitutes an alternative activation pathway for a subset of CD8+ T cells. European Journal of Immunology. 35: 2071-80
Gumá M, Busch L, Salazar-Fontana L, et al. (2005) Correction: The CD94/NKG2C killer lectin-like receptor constitutes an alternative activation pathway for a subset of CD8+ T cells (Vol. 35 (7) 2005, DOI 10.1002/eji.200425843) European Journal of Immunology. 35: 2259-2259
Stunz LL, Busch LK, Munroe ME, et al. (2004) Expression of the cytoplasmic tail of LMP1 in mice induces hyperactivation of B lymphocytes and disordered lymphoid architecture. Immunity. 21: 255-66
Bishop GA, Busch LK. (2002) Molecular mechanisms of B-lymphocyte transformation by Epstein-Barr virus. Microbes and Infection / Institut Pasteur. 4: 853-7
Busch LK, Bishop GA. (2001) Multiple carboxyl-terminal regions of the EBV oncoprotein, latent membrane protein 1, cooperatively regulate signaling to B lymphocytes via TNF receptor-associated factor (TRAF)-dependent and TRAF-independent mechanisms. Journal of Immunology (Baltimore, Md. : 1950). 167: 5805-13
Bishop GA, Ramirez LM, Baccam M, et al. (2001) The immune response modifier resiquimod mimics CD40-induced B cell activation. Cellular Immunology. 208: 9-17
Busch LK, Bishop GA. (1999) The EBV transforming protein, latent membrane protein 1, mimics and cooperates with CD40 signaling in B lymphocytes. Journal of Immunology (Baltimore, Md. : 1950). 162: 2555-61
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