Jingtao Lu, Ph.D.
Affiliations: | 2012 | Biochemistry and Molecular Biology - Environmental Toxicology | Michigan State University, East Lansing, MI |
Area:
ToxicologyGoogle:
"Jingtao Lu"Parents
Sign in to add mentor| Patricia E. Ganey | grad student | 2012 | Michigan State | |
| (Mechanisms of hepatotoxic interaction of amiodarone with inflammatory stress---A model of idiosyncratic, drug-induced liver injury.) | ||||
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Publications
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| Lu J, Roth RA, Malle E, et al. (2013) Roles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide. Toxicological Sciences : An Official Journal of the Society of Toxicology. 136: 51-62 |
| Lu J, Miyakawa K, Roth RA, et al. (2013) Tumor necrosis factor-alpha potentiates the cytotoxicity of amiodarone in Hepa1c1c7 cells: roles of caspase activation and oxidative stress. Toxicological Sciences : An Official Journal of the Society of Toxicology. 131: 164-78 |
| Lu J, Jones AD, Harkema JR, et al. (2012) Amiodarone exposure during modest inflammation induces idiosyncrasy-like liver injury in rats: role of tumor necrosis factor-alpha. Toxicological Sciences : An Official Journal of the Society of Toxicology. 125: 126-33 |
| Deng X, Lu J, Lehman-McKeeman LD, et al. (2008) p38 mitogen-activated protein kinase-dependent tumor necrosis factor-alpha-converting enzyme is important for liver injury in hepatotoxic interaction between lipopolysaccharide and ranitidine. The Journal of Pharmacology and Experimental Therapeutics. 326: 144-52 |
| Deng X, Luyendyk JP, Zou W, et al. (2007) Neutrophil interaction with the hemostatic system contributes to liver injury in rats cotreated with lipopolysaccharide and ranitidine. The Journal of Pharmacology and Experimental Therapeutics. 322: 852-61 |