David M. Terrian

Affiliations: 
East Carolina University, Greenville, NC, United States 
Area:
Cell Biology, Molecular Biology
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"David Terrian"
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Publications

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Chappell WH, Abrams SL, Lertpiriyapong K, et al. (2015) Novel roles of androgen receptor, epidermal growth factor receptor, TP53, regulatory RNAs, NF-kappa-B, chromosomal translocations, neutrophil associated gelatinase, and matrix metalloproteinase-9 in prostate cancer and prostate cancer stem cells. Advances in Biological Regulation
Chappell WH, Lehmann BD, Terrian DM, et al. (2012) p53 expression controls prostate cancer sensitivity to chemotherapy and the MDM2 inhibitor Nutlin-3. Cell Cycle (Georgetown, Tex.). 11: 4579-88
Wescott GG, Manring CM, Terrian DM. (2012) Translocation assays of protein kinase C activation. Methods in Molecular Medicine. 22: 125-32
Zeng Y, Abdallah A, Lu JP, et al. (2009) delta-Catenin promotes prostate cancer cell growth and progression by altering cell cycle and survival gene profiles. Molecular Cancer. 8: 19
Lehmann BD, Paine MS, Brooks AM, et al. (2008) Senescence-associated exosome release from human prostate cancer cells. Cancer Research. 68: 7864-71
Lee JT, Lehmann BD, Terrian DM, et al. (2008) Targeting prostate cancer based on signal transduction and cell cycle pathways. Cell Cycle (Georgetown, Tex.). 7: 1745-62
McCubrey JA, Sokolosky ML, Lehmann BD, et al. (2008) Alteration of Akt activity increases chemotherapeutic drug and hormonal resistance in breast cancer yet confers an achilles heel by sensitization to targeted therapy. Advances in Enzyme Regulation. 48: 113-35
Lehmann BD, Brooks AM, Paine MS, et al. (2008) Distinct roles for p107 and p130 in Rb-independent cellular senescence. Cell Cycle (Georgetown, Tex.). 7: 1262-8
Steelman LS, Navolanic PM, Sokolosky ML, et al. (2008) Suppression of PTEN function increases breast cancer chemotherapeutic drug resistance while conferring sensitivity to mTOR inhibitors. Oncogene. 27: 4086-95
Lehmann BD, McCubrey JA, Terrian DM. (2007) Radiosensitization of prostate cancer by priming the wild-type p53-dependent cellular senescence pathway. Cancer Biology & Therapy. 6: 1165-70
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