William C. Skarnes
Affiliations: | University of California, Berkeley, Berkeley, CA, United States |
Area:
Molecular Biology, Cell Biology, Animal Physiology BiologyGoogle:
"William Skarnes"Children
Sign in to add trainee| Jeremy F. Reiter | grad student | UC Berkeley | |
| Joel A. Zupicich | grad student | 2001 | UC Berkeley |
| Eun-Jun Yun | grad student | 2004 | UC Berkeley |
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Publications
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| Liu Z, Tan Y, Flynn WF, et al. (2025) HAND1, partially mediated through ape-specific LTR binding, is essential for human extra-embryonic mesenchyme derivation from iPSCs. Cell Reports. 44: 115568 |
| Adli M, Przybyla L, Burdett T, et al. (2025) MorPhiC Consortium: towards functional characterization of all human genes. Nature. 638: 351-359 |
| Elrick H, Peterson KA, Willis BJ, et al. (2024) Impact of essential genes on the success of genome editing experiments generating 3313 new genetically engineered mouse lines. Scientific Reports. 14: 22626 |
| Funes S, Jung J, Gadd DH, et al. (2024) Expression of ALS-PFN1 impairs vesicular degradation in iPSC-derived microglia. Nature Communications. 15: 2497 |
| Ryan M, McDonough JA, Ward ME, et al. (2024) Large structural variants in KOLF2.1J are unlikely to compromise neurological disease modeling. Cell Stem Cell. 31: 290-291 |
| Ryan M, McDonough JA, Ward ME, et al. (2024) Large structural variants in KOLF2.1J are unlikely to compromise neurological disease modelling. Biorxiv : the Preprint Server For Biology |
| Nurieva W, Ivanova E, Chehab S, et al. (2023) Generation of four gene-edited human induced pluripotent stem cell lines with mutations in the ATM gene to model Ataxia-Telangiectasia. Stem Cell Research. 73: 103247 |
| Funes S, Gadd DH, Mosqueda M, et al. (2023) Expression of ALS-PFN1 impairs vesicular degradation in iPSC-derived microglia. Biorxiv : the Preprint Server For Biology |
| Pantazis CB, Yang A, Lara E, et al. (2022) A reference human induced pluripotent stem cell line for large-scale collaborative studies. Cell Stem Cell. 29: 1685-1702.e22 |
| Lin YC, Kumar MS, Ramesh N, et al. (2021) Interactions between ALS-linked FUS and nucleoporins are associated with defects in the nucleocytoplasmic transport pathway. Nature Neuroscience |