Michael Klagsbrun
Affiliations: | Harvard University, Cambridge, MA, United States |
Area:
Cell Biology, Molecular BiologyGoogle:
"Michael Klagsbrun"Children
Sign in to add trainee| Jay W. Harper | grad student | 2000 | Harvard |
| Jane Azizkhan-Clifford | post-doc | (Microtree) |
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Publications
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| Wedel J, Nakayama H, Kochupurakkal NM, et al. (2016) The intragraft microenvironment as a central determinant of chronic rejection or local immunoregulation/tolerance. Current Opinion in Organ Transplantation |
| Mucka P, Levonyak N, Geretti E, et al. (2016) Inflammation and Lymphedema Are Exacerbated and Prolonged by Neuropilin 2 Deficiency. The American Journal of Pathology |
| Boneschansker L, Nakayama H, Eisenga M, et al. (2016) Netrin-1 Augments Chemokinesis in CD4+ T Cells In Vitro and Elicits a Proinflammatory Response In Vivo. Journal of Immunology (Baltimore, Md. : 1950) |
| Shim JW, Sandlund J, Hameed MQ, et al. (2016) Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus. Scientific Reports. 6: 26794 |
| Yoshida A, Shimizu A, Asano H, et al. (2015) VEGF-A/NRP1 stimulates GIPC1 and Syx complex formation to promote RhoA activation and proliferation in skin cancer cells. Biology Open. 4: 1063-76 |
| Nakayama H, Bruneau S, Kochupurakkal N, et al. (2015) Regulation of mTOR Signaling by Semaphorin 3F-Neuropilin 2 Interactions In Vitro and In Vivo. Scientific Reports. 5: 11789 |
| Nakayama H, Huang L, Kelly RP, et al. (2015) Infantile hemangioma-derived stem cells and endothelial cells are inhibited by class 3 semaphorins. Biochemical and Biophysical Research Communications |
| Hofmann NA, Yang J, Trauger SA, et al. (2015) The GPR 55 agonist, L-α-lysophosphatidylinositol, mediates ovarian carcinoma cell-induced angiogenesis. British Journal of Pharmacology |
| Adapala RK, Thoppil RJ, Ghosh K, et al. (2015) Activation of mechanosensitive ion channel TRPV4 normalizes tumor vasculature and improves cancer therapy. Oncogene |
| Boscolo E, Coma S, Luks VL, et al. (2015) AKT hyper-phosphorylation associated with PI3K mutations in lymphatic endothelial cells from a patient with lymphatic malformation. Angiogenesis. 18: 151-62 |