Wayne V. Vedeckis
Affiliations: | Louisiana State University Health Sciences Center, New Orleans, LA, United States |
Area:
Cell Biology, Molecular Biology, OncologyGoogle:
"Wayne Vedeckis"
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Publications
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| Miller AL, Geng C, Golovko G, et al. (2014) Epigenetic alteration by DNA-demethylating treatment restores apoptotic response to glucocorticoids in dexamethasone-resistant human malignant lymphoid cells. Cancer Cell International. 14: 35 |
| Sarvaiya PJ, Schwartz JR, Hernandez CP, et al. (2012) Role of c-Myb in the survival of pre B-cell acute lymphoblastic leukemia and leukemogenesis. American Journal of Hematology. 87: 969-76 |
| Schwartz JR, Sarvaiya PJ, Leiva LE, et al. (2012) A facile, branched DNA assay to quantitatively measure glucocorticoid receptor auto-regulation in T-cell acute lymphoblastic leukemia. Chinese Journal of Cancer. 31: 381-91 |
| Sarvaiya PJ, Schwartz JR, Geng CD, et al. (2012) c-Myb interacts with the glucocorticoid receptor and regulates its level in pre-B-acute lymphoblastic leukemia cells. Molecular and Cellular Endocrinology. 361: 124-32 |
| Geng CD, Vedeckis WV. (2011) A new, lineage specific, autoup-regulation mechanism for human glucocorticoid receptor gene expression in 697 pre-B-acute lymphoblastic leukemia cells. Molecular Endocrinology (Baltimore, Md.). 25: 44-57 |
| Geng CD, Vedeckis WV. (2010) Use of recombinant cell-permeable small peptides to modulate glucocorticoid sensitivity of acute lymphoblastic leukemia cells. Biochemistry. 49: 8892-901 |
| Schwartz JR, Sarvaiya PJ, Vedeckis WV. (2010) Glucocorticoid receptor knock down reveals a similar apoptotic threshold but differing gene regulation patterns in T-cell and pre-B-cell acute lymphoblastic leukemia. Molecular and Cellular Endocrinology. 320: 76-86 |
| Geng CD, Schwartz JR, Vedeckis WV. (2008) A conserved molecular mechanism is responsible for the auto-up-regulation of glucocorticoid receptor gene promoters. Molecular Endocrinology (Baltimore, Md.). 22: 2624-42 |
| Geng CD, Vedeckis WV. (2005) c-Myb and members of the c-Ets family of transcription factors act as molecular switches to mediate opposite steroid regulation of the human glucocorticoid receptor 1A promoter. The Journal of Biological Chemistry. 280: 43264-71 |
| Geng CD, Pedersen KB, Nunez BS, et al. (2005) Human glucocorticoid receptor alpha transcript splice variants with exon 2 deletions: evidence for tissue- and cell type-specific functions. Biochemistry. 44: 7395-405 |