Erik S. Knudsen
Affiliations: | University of Cincinnati, Cincinnati, OH |
Area:
Cell Biology, Molecular BiologyGoogle:
"Erik Knudsen"Children
Sign in to add trainee| Matthew W. Strobeck | grad student | 2002 | University of Cincinnati |
| Steven P. Angus | grad student | 2003 | University of Cincinnati |
| Hasan Siddiqui | grad student | 2006 | University of Cincinnati |
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Publications
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| Kumarasamy V, Wang J, Frangou C, et al. (2024) The extracellular niche and tumor microenvironment enhance KRAS inhibitor efficacy in pancreatic cancer. Cancer Research |
| Knudsen ES, Witkiewicz AK, Rubin SM. (2023) Cancer takes many paths through G1/S. Trends in Cell Biology |
| Kumarasamy V, Gao Z, Zhao B, et al. (2023) PROTAC-mediated CDK degradation differentially impacts cancer cell cycles due to heterogeneity in kinase dependencies. British Journal of Cancer |
| Kumarasamy V, Frangou C, Wang J, et al. (2023) Pharmacologically targeting KRAS in PDAC models: tumor cell intrinsic and extrinsic impact. Biorxiv : the Preprint Server For Biology |
| Witkiewicz AK, Kumarasamy V, Sanidas I, et al. (2022) Cancer cell cycle dystopia: heterogeneity, plasticity, and therapy. Trends in Cancer |
| Knudsen ES, Kumarasamy V, Nambiar R, et al. (2022) CDK/cyclin dependencies define extreme cancer cell-cycle heterogeneity and collateral vulnerabilities. Cell Reports. 38: 110448 |
| Kumarasamy V, Vail P, Nambiar R, et al. (2020) Functional determinants of cell-cycle plasticity and sensitivity to CDK4/6 inhibition. Cancer Research |
| Braden WA, Lenihan JM, Lan Z, et al. (2020) Retraction for Braden et al., "Distinct Action of the Retinoblastoma Pathway on the DNA Replication Machinery Defines Specific Roles for Cyclin-Dependent Kinase Complexes in Prereplication Complex Assembly and S-Phase Progression". Molecular and Cellular Biology. 40 |
| Roberts PJ, Kumarasamy V, Witkiewicz AK, et al. (2020) Chemotherapy and CDK4/6 inhibitors: Unexpected bedfellows. Molecular Cancer Therapeutics |
| Knudsen ES, Nambiar R, Rosario SR, et al. (2020) Pan-cancer molecular analysis of the RB tumor suppressor pathway. Communications Biology. 3: 158 |