Jonathan Reichner
Affiliations: | Brown University, Providence, RI |
Area:
Immunology, Cell BiologyGoogle:
"Jonathan Reichner"Children
Sign in to add trainee| Liz M. Lavigne | grad student | 2004 | Brown |
| Brian W. LeBlanc | grad student | 2007 | Brown |
| Courtni T. Newsome | grad student | 2010 | Brown |
| Xian M. O'Brien | grad student | 2010 | Brown |
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Publications
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| Wilson ZS, Witt H, Hazlett L, et al. (2020) Context-Dependent Role of Vinculin in Neutrophil Adhesion, Motility and Trafficking. Scientific Reports. 10: 2142 |
| Dickinson CM, LeBlanc BW, Edhi MM, et al. (2018) Leukadherin-1 ameliorates endothelial barrier damage mediated by neutrophils from critically ill patients. Journal of Intensive Care. 6: 19 |
| Harman MW, Franck C, Reichner J. (2018) Four Dimensional Traction Measurements of Chemotactic Neutrophils in Hydrogels Biophysical Journal. 114: 324a |
| Johnson CM, O'Brien XM, Byrd AS, et al. (2016) Integrin Cross-Talk Regulates the Human Neutrophil Response to Fungal β-Glucan in the Context of the Extracellular Matrix: A Prominent Role for VLA3 in the Antifungal Response. Journal of Immunology (Baltimore, Md. : 1950) |
| O'Brien XM, Biron BM, Reichner JS. (2016) Consequences of extracellular trap formation in sepsis. Current Opinion in Hematology |
| O'Brien XM, Reichner JS. (2016) Neutrophil Integrins and Matrix Ligands and NET Release. Frontiers in Immunology. 7: 363 |
| Biron BM, Chung CS, O'Brien XM, et al. (2016) Cl-Amidine Prevents Histone 3 Citrullination and Neutrophil Extracellular Trap Formation, and Improves Survival in a Murine Sepsis Model. Journal of Innate Immunity |
| Byrd AS, O'Brien XM, Laforce-Nesbitt SS, et al. (2015) NETosis in Neonates: Evidence of a Reactive Oxygen Species-Independent Pathway in Response to Fungal Challenge. The Journal of Infectious Diseases |
| Loosley AJ, O'Brien XM, Reichner JS, et al. (2015) Describing directional cell migration with a characteristic directionality time. Plos One. 10: e0127425 |
| Magruder HT, Quinn JA, Schwartzbauer JE, et al. (2014) The G protein-coupled estrogen receptor-1, GPER-1, promotes fibrillogenesis via a Shc-dependent pathway resulting in anchorage-independent growth. Hormones & Cancer. 5: 390-404 |