Clinton D. Lothrop

Affiliations: 
Auburn University, Auburn, AL, United States 
Area:
Veterinary Science Biology, Molecular Biology
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"Clinton Lothrop"
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Publications

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Niemeyer GP, Herzog RW, Mount J, et al. (2009) Long-term correction of inhibitor-prone hemophilia B dogs treated with liver-directed AAV2-mediated factor IX gene therapy. Blood. 113: 797-806
Tillson M, Niemeyer GP, Welch JA, et al. (2006) Hematopoietic chimerism induces renal and skin allograft tolerance in DLA-identical dogs. Experimental Hematology. 34: 1759-70
Broaddus KD, Tillson DM, Lenz SD, et al. (2006) Renal allograft histopathology in dog leukocyte antigen mismatched dogs after renal transplantation. Veterinary Surgery : Vs. 35: 125-35
Meng R, Upchurch D, Niemeyer GP, et al. (2006) Increased Unfolded Protein Response in Hematopoietic Cells from Cyclic Hematopoietic Dogs. Blood. 108: 4185-4185
Niemeyer GP, Herzog RW, Mount JD, et al. (2006) Six-Year Follow-Up of Inhibitor Prone Hemophilia B Dogs Treated with Muscle and Liver-Directed AAV2 Mediated Factor IX Gene Therapy. Blood. 108: 3282-3282
Niemeyer GP, Boudreaux MK, Goodman-Martin SA, et al. (2003) Correction of a large animal model of type I Glanzmann's thrombasthenia by nonmyeloablative bone marrow transplantation. Experimental Hematology. 31: 1357-62
Takatu A, Nash RA, Zaucha JM, et al. (2003) Adoptive immunotherapy to increase the level of donor hematopoietic chimerism after nonmyeloablative marrow transplantation for severe canine hereditary hemolytic anemia. Biology of Blood and Marrow Transplantation : Journal of the American Society For Blood and Marrow Transplantation. 9: 674-82
Benson KF, Li FQ, Person RE, et al. (2003) Mutations associated with neutropenia in dogs and humans disrupt intracellular transport of neutrophil elastase. Nature Genetics. 35: 90-6
Mount JD, Herzog RW, Tillson DM, et al. (2002) Sustained phenotypic correction of hemophilia B dogs with a factor IX null mutation by liver-directed gene therapy. Blood. 99: 2670-6
Herzog RW, Mount JD, Arruda VR, et al. (2001) Muscle-directed gene transfer and transient immune suppression result in sustained partial correction of canine hemophilia B caused by a null mutation Molecular Therapy. 4: 192-200
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