Kevin J. Trouba, Ph.D.

Affiliations: 
2000 University of Nebraska Medical Center, Omaha, NE, United States 
Area:
Pharmacology, Oncology
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"Kevin Trouba"

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Roseann L. Vorce grad student 2000 University of Nebraska Medical Center
 (Deregulation of growth factor-induced signaling events following sodium arsenite exposure.)
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Publications

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Patterson R, Vega L, Trouba K, et al. (2004) Arsenic-induced alterations in the contact hypersensitivity response in Balb/c mice. Toxicology and Applied Pharmacology. 198: 434-43
Trouba KJ, Germolec DR. (2004) Micromolar concentrations of sodium arsenite induce cyclooxygenase-2 expression and stimulate p42/44 mitogen-activated protein kinase phosphorylation in normal human epidermal keratinocytes. Toxicological Sciences : An Official Journal of the Society of Toxicology. 79: 248-57
Trouba KJ, Geisenhoffer KM, Germolec DR. (2002) Sodium arsenite-induced stress-related gene expression in normal human epidermal, HaCaT, and HEL30 keratinocytes. Environmental Health Perspectives. 110: 761-6
Hamadeh HK, Trouba KJ, Amin RP, et al. (2002) Coordination of altered DNA repair and damage pathways in arsenite-exposed keratinocytes. Toxicological Sciences : An Official Journal of the Society of Toxicology. 69: 306-16
Trouba KJ, Wauson EM, Vorce RL. (2000) Sodium arsenite inhibits terminal differentiation of murine C3H 10T1/2 preadipocytes. Toxicology and Applied Pharmacology. 168: 25-35
Trouba KJ, Wauson EM, Vorce RL. (2000) Sodium arsenite-induced dysregulation of proteins involved in proliferative signaling. Toxicology and Applied Pharmacology. 164: 161-70
Trouba KJ, Glanzer JG, Vorce RL. (1999) Wild-type and Ras-transformed fibroblasts display differential mitogenic responses to transient sodium arsenite exposure. Toxicological Sciences : An Official Journal of the Society of Toxicology. 50: 72-81
Liao L, Trouba KJ, Vorce RL. (1998) Determinants of species- and placenta-specific expression of p100 GAP. Placenta. 19: 225-30
Trouba KJ, Liao L, Vorce RL. (1996) Compensatory modulation of GAP activity in response to oncogenic stimulation. Cancer Letters. 109: 211-5
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