Yihru Fannjiang, Ph.D.
Affiliations: | 2004 | Johns Hopkins University, Baltimore, MD |
Area:
Molecular Biology, Neuroscience Biology, Cell BiologyGoogle:
"Yihru Fannjiang"Parents
Sign in to add mentor| Marie Hardwick | grad student | 2004 | Johns Hopkins | |
| (From yeast to mammals: A functional analysis of Fis-1 and Bak in programmed cell death.) | ||||
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Publications
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| Fannjiang Y, Cheng WC, Lee SJ, et al. (2004) Mitochondrial fission proteins regulate programmed cell death in yeast. Genes & Development. 18: 2785-97 |
| Jonas EA, Hickman JA, Chachar M, et al. (2004) Proapoptotic N-truncated BCL-xL protein activates endogenous mitochondrial channels in living synaptic terminals. Proceedings of the National Academy of Sciences of the United States of America. 101: 13590-5 |
| Seo SY, Chen YB, Ivanovska I, et al. (2004) BAD is a pro-survival factor prior to activation of its pro-apoptotic function. The Journal of Biological Chemistry. 279: 42240-9 |
| Jonas EA, Hoit D, Hickman JA, et al. (2003) Modulation of synaptic transmission by the BCL-2 family protein BCL-xL. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 23: 8423-31 |
| Fannjiang Y, Kim CH, Huganir RL, et al. (2003) BAK alters neuronal excitability and can switch from anti- to pro-death function during postnatal development. Developmental Cell. 4: 575-85 |
| Kerr DA, Larsen T, Cook SH, et al. (2002) BCL-2 and BAX protect adult mice from lethal Sindbis virus infection but do not protect spinal cord motor neurons or prevent paralysis. Journal of Virology. 76: 10393-400 |