Ila Joshi, Ph.D.
Affiliations: | 2008 | Molecular & Cellular Biology | University of Massachusetts, Amherst, Amherst, MA |
Area:
Molecular Biology, ImmunologyGoogle:
"Ila Joshi"Parents
Sign in to add mentor| Barbara A. Osborne | grad student | 2008 | U Mass Amherst | |
| (The role of Notch in regulation of G1 -S progression of cell cycle in T cells.) | ||||
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Publications
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| Hu Y, Zhang Z, Kashiwagi M, et al. (2016) Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia. Genes & Development. 30: 1971-90 |
| Etten RV, Hu Y, Zhang Z, et al. (2016) Central role for ikaros in pre-B cell differentiation and pathogenesis of high-risk B-cell acute lymphoblastic leukemia Experimental Hematology. 44 |
| Bakshi R, Zhang H, Logan R, et al. (2015) Neuroprotective effects of urate are mediated by augmenting astrocytic glutathione synthesis and release. Neurobiology of Disease |
| Jena N, Joshi I, Yoshida T, et al. (2015) Abstract 4202: Targeting focal adhesion kinase is a novel approach to therapy of high-risk, Ikaros-mutant acute B-cell lymphoblastic leukemia Cancer Research. 75: 4202-4202 |
| Joshi I, Yoshida T, Jena N, et al. (2014) Loss of Ikaros DNA-binding function confers integrin-dependent survival on pre-B cells and progression to acute lymphoblastic leukemia. Nature Immunology. 15: 294-304 |
| Joshi I, Jena N, Yoshida T, et al. (2014) Focal Adhesion Kinase Inhibitors Reverse the Stromal Adhesion Phenotype of Ikaros-Mutant B-ALL, Induce Apopotosis, and Synergize with ABL1 Tyrosine Kinase Inhibitors: A New Paradigm for Pathogenesis and Therapy of High-Risk B-ALL Blood. 124: 285-285 |
| Jena N, Joshi I, Yoshida T, et al. (2013) Ikaros Mutation Confers Integrin-Dependent Survival Of Pre-B Cells and Progression To Acute Lymphoblastic Leukemia Blood. 122: 1259-1259 |
| Joshi I, Minter LM, Telfer J, et al. (2009) Notch signaling mediates G1/S cell-cycle progression in T cells via cyclin D3 and its dependent kinases. Blood. 113: 1689-98 |
| Izadi H, Motameni AT, Bates TC, et al. (2007) c-Jun N-terminal kinase 1 is required for Toll-like receptor 1 gene expression in macrophages. Infection and Immunity. 75: 5027-34 |