Benjamin L. Unger, Ph.D.
Affiliations: | 2009 | Biological Sciences | State University of New York at Binghamton, Vestal, NY, United States |
Area:
Cell BiologyGoogle:
"Benjamin Unger"Parents
Sign in to add mentor| Dennis W. McGee | grad student | 2009 | SUNY Binghamton | |
| (The role of growth factors and Rho kinase (ROCK) in the regulation of IL-1 mediated pro-inflammatory cytokine production in intestinal epithelial cells.) | ||||
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| Ganesan S, Pham D, Jing Y, et al. (2016) TLR2 Activation Limits Rhinovirus-Stimulated CXCL-10 by Attenuating IRAK-1-Dependent IL-33 Receptor Signaling in Human Bronchial Epithelial Cells. Journal of Immunology (Baltimore, Md. : 1950) |
| Ganesan S, Unger BL, Comstock AT, et al. (2013) Aberrantly activated EGFR contributes to enhanced IL-8 expression in COPD airways epithelial cells via regulation of nuclear FoxO3A. Thorax. 68: 131-41 |
| Rafferty BJ, Unger BL, Perey AC, et al. (2012) A novel role for the Rho-associated kinase, ROCK, in IL-1-stimulated intestinal epithelial cell responses. Cellular Immunology. 280: 148-55 |
| Unger BL, McGee DW. (2011) Hepatocyte growth factor and keratinocyte growth factor enhance IL-1-induced IL-8 secretion through different mechanisms in Caco-2 epithelial cells In Vitro Cellular and Developmental Biology - Animal. 47: 173-181 |
| Grygas J, Steiger N, LeSeur CL, et al. (2007) Hepatocyte growth factor enhances IL-1beta stimulated IL-8 secretion by Caco-2 epithelial cells. In Vitro Cellular & Developmental Biology. Animal. 43: 147-52 |