Jamie L. Fornsaglio, Ph.D.

Affiliations: 
2004 The George Washington University, Washington, DC, United States 
Area:
Oncology, Molecular Biology, Pharmacology
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"Jamie Fornsaglio"

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Steven R. Patierno grad student 2004 The George Washington University
 (The mechanisms involved in the repair of hexavalent chromium-induced DNA damage: Role of recombinational repair.)
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Publications

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Choi YJ, Li H, Son MY, et al. (2014) Deletion of individual Ku subunits in mice causes an NHEJ-independent phenotype potentially by altering apurinic/apyrimidinic site repair. Plos One. 9: e86358
Momcilovic O, Knobloch L, Fornsaglio J, et al. (2010) DNA damage responses in human induced pluripotent stem cells and embryonic stem cells. Plos One. 5: e13410
Jelezcova E, Trivedi RN, Wang XH, et al. (2010) Parp1 activation in mouse embryonic fibroblasts promotes Pol beta-dependent cellular hypersensitivity to alkylation damage. Mutation Research. 686: 57-67
Pritchard DE, Ceryak S, Ramsey KE, et al. (2005) Resistance to apoptosis, increased growth potential, and altered gene expression in cells that survived genotoxic hexavalent chromium [Cr(VI)] exposure. Molecular and Cellular Biochemistry. 279: 169-81
Fornsaglio JL, O'Brien TJ, Patierno SR. (2005) Differential impact of ionic and coordinate covalent chromium (Cr)-DNA binding on DNA replication. Molecular and Cellular Biochemistry. 279: 149-55
Trivedi RN, Almeida KH, Fornsaglio JL, et al. (2005) The role of base excision repair in the sensitivity and resistance to temozolomide-mediated cell death. Cancer Research. 65: 6394-400
O'Brien TJ, Fornsaglio JL, Ceryak S, et al. (2002) Effects of hexavalent chromium on the survival and cell cycle distribution of DNA repair-deficient S. cerevisiae. Dna Repair. 1: 617-27
Vilcheck SK, O'Brien TJ, Pritchard DE, et al. (2002) Fanconi anemia complementation group A cells are hypersensitive to chromium(VI)-induced toxicity. Environmental Health Perspectives. 110: 773-7
Pritchard DE, Ceryak S, Ha L, et al. (2001) Mechanism of apoptosis and determination of cellular fate in chromium(VI)-exposed populations of telomerase-immortalized human fibroblasts. Cell Growth & Differentiation : the Molecular Biology Journal of the American Association For Cancer Research. 12: 487-96
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