William B. Guggino
Affiliations: | Johns Hopkins University, Baltimore, MD |
Area:
Cell BiologyGoogle:
"William Guggino"Children
Sign in to add traineeSang-Ho Kwon | grad student | 2005 | Johns Hopkins |
Yinghong Wang | grad student | 2005 | Johns Hopkins |
Netty G. Santoso | grad student | 2011 | Johns Hopkins |
Ha W. Lee | grad student | 2012 | Johns Hopkins |
Deepali N. Tukaye | grad student | 2012 | Johns Hopkins |
Emily Anne Smith Bergbower | grad student | 2015 | Johns Hopkins Medical School |
Owen M. Woodward | post-doc | Johns Hopkins Medical School (Neurotree) |
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Publications
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Yanda MK, Guggino WB, Cebotaru L. (2019) A new role for heat shock factor 27 in the pathophysiology of Clostridium difficile toxin B. American Journal of Physiology. Gastrointestinal and Liver Physiology |
Bergbower EAS, Sabirzhanova I, Boinot C, et al. (2019) Restoration of F508-del Function by Transcomplementation: The Partners Meet in the Endoplasmic Reticulum. Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology. 52: 1267-1279 |
Sabirzhanova I, Boinot C, Guggino WB, et al. (2018) Syntaxin 8 and the Endoplasmic Reticulum Processing of ΔF508-CFTR. Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology. 51: 1489-1499 |
Yanda MK, Liu Q, Cebotaru V, et al. (2018) Role of calcium in adult onset polycystic kidney disease. Cellular Signalling |
Liu Q, Sabirzhanova I, Yanda MK, et al. (2018) Rescue of CFTR NBD2 mutants N1303K and S1235R is influenced by the functioning of the autophagosome. Journal of Cystic Fibrosis : Official Journal of the European Cystic Fibrosis Society |
Krishnan V, Park SA, Shin SS, et al. (2018) Wireless control of cellular function by activation of a novel protein responsive to electromagnetic fields. Scientific Reports. 8: 8764 |
Bergbower E, Boinot C, Sabirzhanova I, et al. (2018) The CFTR-Associated Ligand Arrests the Trafficking of the Mutant ΔF508 CFTR Channel in the ER Contributing to Cystic Fibrosis Cellular Physiology and Biochemistry. 45: 639-655 |
Yanda MK, Liu Q, Cebotaru V, et al. (2017) Histone deacetylase 6 Inhibition reduces cysts by decreasing via cAMP and Ca2+ in knockout mouse models of polycystic kidney disease. The Journal of Biological Chemistry |
Wang H, Cebotaru L, Lee HW, et al. (2016) CFTR Controls the Activity of NF-κB by Enhancing the Degradation of TRADD. Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology. 40: 1063-1078 |
Cebotaru L, Liu Q, Yanda M, et al. (2016) Inhibition of histone deacetylase 6 activity reduces cyst growth in polycystic kidney disease. Kidney International |