Richard B. Marchase

Affiliations: 
University of Alabama, Birmingham, Birmingham, AL, United States 
Area:
Cell Biology, Physiology Biology
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"Richard Marchase"

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Stephen Roth grad student (Anatomy Tree)
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Publications

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Collins HE, He L, Zou L, et al. (2014) Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function. American Journal of Physiology. Heart and Circulatory Physiology. 306: H1231-9
Taylor EW, Wang K, Nelson AR, et al. (2014) O-GlcNAcylation of AMPA receptor GluA2 is associated with a novel form of long-term depression at hippocampal synapses. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 10-21
Collins HE, Zhu-Mauldin X, Marchase RB, et al. (2013) STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology. American Journal of Physiology. Heart and Circulatory Physiology. 305: H446-58
Zhu-Mauldin X, Marsh SA, Zou L, et al. (2012) Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytes. The Journal of Biological Chemistry. 287: 39094-106
Zou L, Zhu-Mauldin X, Marchase RB, et al. (2012) Glucose deprivation-induced increase in protein O-GlcNAcylation in cardiomyocytes is calcium-dependent. The Journal of Biological Chemistry. 287: 34419-31
Chatham JC, Marchase RB. (2010) Protein O-GlcNAcylation: A critical regulator of the cellular response to stress. Current Signal Transduction Therapy. 5: 49-59
Nöt LG, Brocks CA, Vámhidy L, et al. (2010) Increased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in rats. Critical Care Medicine. 38: 562-71
Chatham JC, Marchase RB. (2010) The role of protein O-linked beta-N-acetylglucosamine in mediating cardiac stress responses. Biochimica Et Biophysica Acta. 1800: 57-66
Zou L, Yang S, Champattanachai V, et al. (2009) Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling. American Journal of Physiology. Heart and Circulatory Physiology. 296: H515-23
Champattanachai V, Marchase RB, Chatham JC. (2008) Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2. American Journal of Physiology. Cell Physiology. 294: C1509-20
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