Patricia V. Schoenlein
Affiliations: | Medical College of Georgia, Augusta, GA, United States |
Area:
Oncology, Cell Biology, Molecular BiologyGoogle:
"Patricia Schoenlein"
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Publications
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| McGrath MK, Abolhassani A, Guy L, et al. (2024) Autophagy and senescence facilitate the development of antiestrogen resistance in ER positive breast cancer. Frontiers in Endocrinology. 15: 1298423 |
| Elshazly AM, Sinanian MM, Neely V, et al. (2023) BRD4 Inhibition as a Strategy to Prolong the Response to Standard of Care in Estrogen Receptor-Positive Breast Cancer. Cancers. 15 |
| Hagan ML, Mander S, Joseph C, et al. (2022) Upregulation of the EGFR/MEK1/MAPK1/2 signaling axis as a mechanism of resistance to antiestrogen‑induced BimEL dependent apoptosis in ER breast cancer cells. International Journal of Oncology. 62 |
| Finnegan RM, Elshazly AM, Schoenlein PV, et al. (2022) Therapeutic Potential for Targeting Autophagy in ER+ Breast Cancer. Cancers. 14 |
| Herrington K, Joseph C, Abdulmoneim S, et al. (2020) Abstract 1231: Targeting JNK1/2 induces a potent cytostatic response in estrogen receptor expressing breast cancer cells, with induction of autophagy and senescence as measurable outcomes Cancer Research. 80: 1231-1231 |
| Meghil MM, Tawfik OK, Elashiry M, et al. (2019) Disruption of Immune Homeostasis in Human Dendritic Cells via Regulation of Autophagy and Apoptosis by . Frontiers in Immunology. 10: 2286 |
| Munagala R, Joseph C, Liu A, et al. (2017) Abstract 3318: A critical role for c-Jun N-terminal kinase in autophagy and cell survival of breast cancer cells Cancer Research. 77: 3318-3318 |
| Liu F, Li X, Lu C, et al. (2016) Ceramide activates lysosomal cathepsin B and cathepsin D to attenuate autophagy and induces ER stress to suppress myeloid-derived suppressor cells. Oncotarget |
| Conger J, Manning M, Anosike K, et al. (2016) Abstract 4672: Targeting MEK/MAPK1/2in vivoto eradicate antiestrogen resistance Cancer Research. 76: 4672-4672 |
| Manning M, Takhar S, Periyasamy-Thandavan S, et al. (2015) Abstract 1005: Dual targeting of MEK/MAPK1/2 and pro-survival autophagy to optimize antiestrogen treatment toward the eradication of antiestrogen resistant breast cancer Cancer Research. 75: 1005-1005 |