Jun Ninomiya-Tsuji
Affiliations: | North Carolina State University, Raleigh, NC |
Area:
Cell Biology, ToxicologyGoogle:
"Jun Ninomiya-Tsuji"
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Publications
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| López-Pérez W, Sai K, Sakamachi Y, et al. (2021) TAK1 inhibition elicits mitochondrial ROS to block intracellular bacterial colonization. Proceedings of the National Academy of Sciences of the United States of America. 118 |
| Sai K, Parsons C, House JS, et al. (2019) Necroptosis mediators RIPK3 and MLKL suppress intracellular replication independently of host cell killing. The Journal of Cell Biology |
| Sung Hsieh HH, Agarwal S, Cholok DJ, et al. (2019) Coordinating Tissue Regeneration through TGF-β Activated Kinase 1 (TAK1) In-activation and Re-activation. Stem Cells (Dayton, Ohio) |
| Liu X, Hayano S, Pan H, et al. (2018) Compound mutations in Bmpr1a and Tak1 synergize facial deformities via increased cell death. Genesis (New York, N.Y. : 2000) |
| Mihaly SR, Sakamachi Y, Ninomiya-Tsuji J, et al. (2017) Erratum: Noncanonical cell death program independent of caspase activation cascade and necroptotic modules is elicited by loss of TGFβ-activated kinase 1. Scientific Reports. 7: 10695 |
| Mihaly SR, Sakamachi Y, Ninomiya-Tsuji J, et al. (2017) Noncanocial cell death program independent of caspase activation cascade and necroptotic modules is elicited by loss of TGFβ-activated kinase 1. Scientific Reports. 7: 2918 |
| Sakamachi Y, Morioka S, Mihaly SR, et al. (2017) TAK1 regulates resident macrophages by protecting lysosomal integrity. Cell Death & Disease. 8: e2598 |
| Hashimoto K, Simmons AN, Kajino-Sakamoto R, et al. (2016) TAK1 regulates the Nrf2 antioxidant system through modulating p62/ SQSTM1. Antioxidants & Redox Signaling |
| Simmons AN, Kajino-Sakamoto R, Ninomiya-Tsuji J. (2016) TAK1 regulates Paneth cell integrity partly through blocking necroptosis. Cell Death & Disease. 7: e2196 |
| Sai K, Morioka S, Takaesu G, et al. (2016) TAK1 determines susceptibility to endoplasmic reticulum stress and hypothalamic leptin resistance. Journal of Cell Science |