Arthur S. Alberts
Affiliations: | Michigan State University, East Lansing, MI |
Area:
Cell Biology, Molecular BiologyGoogle:
"Arthur Alberts"Children
Sign in to add traineeAaron D. DeWard | grad student | 2009 | Michigan State |
Julie Davis Good (Turner) | research scientist | 2010-2016 | Van Andel Research Institute (Physiology Academic Tree) |
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Publications
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Pettee KM, Becker KN, Alberts AS, et al. (2019) Targeting the mDia Formin-Assembled Cytoskeleton Is an Effective Anti-Invasion Strategy in Adult High-Grade Glioma Patient-Derived Neurospheres. Cancers. 11 |
LeCorgne H, Tudosie AM, Lavik K, et al. (2018) Differential Toxicity of mDia Formin-Directed Functional Agonists and Antagonists in Developing Zebrafish. Frontiers in Pharmacology. 9: 340 |
Erasmus JC, Bruche S, Pizarro L, et al. (2016) Defining functional interactions during biogenesis of epithelial junctions. Nature Communications. 7: 13542 |
Damiani D, Goffinet AM, Alberts A, et al. (2016) Lack of Diaph3 relaxes the spindle checkpoint causing the loss of neural progenitors. Nature Communications. 7: 13509 |
Thiam HR, Vargas P, Carpi N, et al. (2016) Perinuclear Arp2/3-driven actin polymerization enables nuclear deformation to facilitate cell migration through complex environments. Nature Communications. 7: 10997 |
Vargas P, Maiuri P, Bretou M, et al. (2016) Corrigendum: Innate control of actin nucleation determines two distinct migration behaviours in dendritic cells. Nature Cell Biology. 18: 234 |
Davis-Turner J, Howard AM, Alberts AS. (2016) Abstract 1867: Activation of RHO-DIAPH signaling impairs RAS-driven exosome biogenesis Cancer Research. 76: 1867-1867 |
Vargas P, Maiuri P, Bretou M, et al. (2015) Innate control of actin nucleation determines two distinct migration behaviours in dendritic cells. Nature Cell Biology |
Arden JD, Lavik KI, Rubinic KA, et al. (2015) Small molecule agonists of mammalian Diaphanous-related (mDia) formins reveal an effective glioblastoma anti-invasion strategy. Molecular Biology of the Cell |
Ercan-Sencicek AG, Jambi S, Franjic D, et al. (2015) Homozygous loss of DIAPH1 is a novel cause of microcephaly in humans. European Journal of Human Genetics : Ejhg. 23: 165-72 |