Michael H. Nathanson
Affiliations: | Yale University, New Haven, CT |
Area:
Cell BiologyGoogle:
"Michael Nathanson"
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Publications
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Rodrigues MA, Gomes DA, Fiorotto R, et al. (2022) Molecular determinants of peri-apical targeting of inositol 1,4,5-trisphosphate receptor type 3 in cholangiocytes. Hepatology Communications |
Lima Filho ACM, França A, Florentino RM, et al. (2020) Inositol 1,4,5-trisphosphate receptor type 3 plays a protective role in hepatocytes during hepatic ischemia-reperfusion injury. Cell Calcium. 91: 102264 |
Gorelick F, Nathanson MH. (2020) TRPV4 helps Piezo1 put the squeeze on pancreatic acinar cells. The Journal of Clinical Investigation |
Perry RJ, Zhang D, Guerra MT, et al. (2020) Glucagon stimulates gluconeogenesis by INSP3R1-mediated hepatic lipolysis. Nature |
Guerra MT, Leite MF, Nathanson MH. (2020) Ca 2+ Signaling in the Liver Liver. 496-508 |
Mangla A, Guerra MT, Nathanson MH. (2019) Type 3 inositol 1,4,5-trisphosphate receptor: A calcium channel for all seasons. Cell Calcium. 85: 102132 |
Zhang B, Li MD, Yin R, et al. (2019) O-GlcNAc transferase suppresses necroptosis and liver fibrosis. Jci Insight. 4 |
de Miranda MC, Rodrigues MA, de Angelis Campos AC, et al. (2019) Epidermal growth factor (EGF) triggers nuclear calcium signaling through the intranuclear phospholipase C delta-4 (PLCδ4). The Journal of Biological Chemistry |
Guerra MT, Florentino RM, Franca A, et al. (2019) Expression of the type 3 InsP receptor is a final common event in the development of hepatocellular carcinoma. Gut |
Ueasilamongkol P, Khamphaya T, Guerra MT, et al. (2019) Type 3 inositol 1,4,5-trisphosphate receptor is increased and enhances malignant properties in cholangiocarcinoma. Hepatology (Baltimore, Md.) |