Ute Moll
Affiliations: | Molecular and Cellular Biology | Stony Brook University, Stony Brook, NY, United States |
Area:
Cell BiologyWebsite:
https://renaissance.stonybrookmedicine.edu/pathology/faculty/mollGoogle:
"Ute Moll"Children
Sign in to add traineeFlaminia M. Talos | grad student | 2006 | SUNY Stony Brook |
Angelina V. Vaseva | grad student | 2011 | SUNY Stony Brook |
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Publications
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Isermann T, Schneider KL, Wegwitz F, et al. (2024) Enhancement of colorectal cancer therapy through interruption of the HSF1-HSP90 axis by p53 activation or cell cycle inhibition. Biorxiv : the Preprint Server For Biology |
Isermann T, Şener ÖÇ, Stender A, et al. (2021) Suppression of HSF1 activity by wildtype p53 creates a driving force for p53 loss-of-heterozygosity. Nature Communications. 12: 4019 |
Klemke L, Fehlau CF, Winkler N, et al. (2021) The Gain-of-Function p53 R248W Mutant Promotes Migration by STAT3 Deregulation in Human Pancreatic Cancer Cells. Frontiers in Oncology. 11: 642603 |
Fagnan A, Bagger FO, Piqué-Borràs MR, et al. (2020) Human erythroleukemia genetics and transcriptomes identify master transcription factors as functional disease drivers. Blood |
Tutuska K, Parrilla-Monge L, Di Cesare E, et al. (2020) Statin as anti-cancer therapy in autochthonous T-lymphomas expressing stabilized gain-of-function mutant p53 proteins. Cell Death & Disease. 11: 274 |
Nemajerova A, Moll UM. (2019) Tissue-specific roles of p73 in development and homeostasis. Journal of Cell Science. 132 |
Meyer G, González-Arnay E, Moll U, et al. (2019) Cajal-Retzius neurons are required for the development of the human hippocampal fissure. Journal of Anatomy |
Nemajerova A, Amelio I, Gebel J, et al. (2018) Non-oncogenic roles of TAp73: from multiciliogenesis to metabolism. Cell Death and Differentiation. 25: 144-153 |
Schulz-Heddergott R, Stark N, Edmunds SJ, et al. (2018) Therapeutic Ablation of Gain-of-Function Mutant p53 in Colorectal Cancer Inhibits Stat3-Mediated Tumor Growth and Invasion. Cancer Cell. 34: 298-314.e7 |
Schulz-Heddergott R, Moll UM. (2018) Gain-of-Function (GOF) Mutant p53 as Actionable Therapeutic Target. Cancers. 10 |