Weihang Chai
Affiliations: | Molecular Biosciences | Washington State University, Pullman, WA, United States |
Area:
Molecular Biology, Cell Biology, GeneticsGoogle:
"Weihang Chai"
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Jaiswal RK, Lei KH, Chastain M, et al. (2023) CaMKK2 and CHK1 phosphorylate human STN1 in response to replication stress to protect stalled forks from aberrant resection. Nature Communications. 14: 7882 |
Nguyen DD, Kim E, Le NT, et al. (2023) Deficiency in mammalian STN1 promotes colon cancer development via inhibiting DNA repair. Science Advances. 9: eadd8023 |
Lei KH, Yang HL, Chang HY, et al. (2021) Crosstalk between CST and RPA regulates RAD51 activity during replication stress. Nature Communications. 12: 6412 |
Chai W, Chastain M, Shiva O, et al. (2021) The Intrinsically Disordered Region in the Human STN1 OB-Fold Domain Is Important for Protecting Genome Stability. Biology. 10 |
Lyu X, Sang PB, Chai W. (2021) CST in maintaining genome stability: Beyond telomeres. Dna Repair. 102: 103104 |
Lyu X, Lei KH, Biak Sang P, et al. (2020) Human CST complex protects stalled replication forks by directly blocking MRE11 degradation of nascent-strand DNA. The Embo Journal. e103654 |
Nguyen DD, Kim EY, Sang PB, et al. (2020) Roles of OB-Fold Proteins in Replication Stress. Frontiers in Cell and Developmental Biology. 8: 574466 |
Lyu X, Chastain M, Chai W. (2019) Genome-wide mapping and profiling of γH2AX binding hotspots in response to different replication stress inducers. Bmc Genomics. 20: 579 |
Jia P, Chai W. (2018) The MLH1 ATPase domain is needed for suppressing aberrant formation of interstitial telomeric sequences. Dna Repair. 65: 20-25 |
Wang Y, Chai W. (2018) Pathogenic CTC1 mutations cause global genome instabilities under replication stress. Nucleic Acids Research |