Marja M. Hurley
Affiliations: | University of Connecticut, Storrs, CT, United States |
Area:
Cell Biology, Endocrinology Biology, AgingGoogle:
"Marja Hurley"
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Publications
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Dutra EH, Chen PJ, Kalajzic Z, et al. (2023) FGF Ligands and Receptors in Osteochondral Tissues of the Temporomandibular Joint in Young and Aging Mice. Cartilage. 19476035231163691 |
Xiao L, Zhou Y, Bokoliya S, et al. (2022) Bone loss is ameliorated by fecal microbiota transplantation through SCFA/GPR41/ IGF1 pathway in sickle cell disease mice. Scientific Reports. 12: 20638 |
Hurley MM, Coffin JD, Doetschman T, et al. (2022) FGF receptor inhibitor BGJ398 partially rescues osteoarthritis-like phenotype in older high molecular weight FGF2 transgenic mice via multiple mechanisms. Scientific Reports. 12: 15968 |
Millington G, Joseph J, Xiao L, et al. (2021) Fibroblast Growth Factor 2 High Molecular Weight Isoforms in Dentoalveolar Mineralization. Calcified Tissue International |
Kuhn LT, Peng T, Gronowicz G, et al. (2021) Endogenous FGF-2 levels impact FGF-2/BMP-2 growth factor delivery dosing in aged murine calvarial bone defects. Journal of Biomedical Materials Research. Part A |
Xiao L, Williams D, Hurley MM. (2020) Inhibition of FGFR Signaling Partially Rescues Osteoarthritis in Mice Overexpressing High Molecular Weight FGF2 Isoforms. Endocrinology. 161 |
Xiao L, Fei Y, Hurley MM. (2018) FGF2 crosstalk with Wnt signaling in mediating the anabolic action of PTH on bone formation. Bone Reports. 9: 136-144 |
Alhamdi J, Jacobs E, Gronowicz G, et al. (2018) Cell Type Influences Local Delivery of Biomolecules from a Bioinspired Apatite Drug Delivery System. Materials (Basel, Switzerland). 11 |
Burt PM, Xiao L, Doetschman T, et al. (2018) Ablation of low-molecular-weight FGF2 isoform accelerates murine osteoarthritis while loss of high-molecular-weight FGF2 isoforms offers protection. Journal of Cellular Physiology |
Coffin JD, Homer-Bouthiette C, Hurley MM. (2018) Fibroblast Growth Factor 2 and Its Receptors in Bone Biology and Disease. Journal of the Endocrine Society. 2: 657-671 |