Harry L. Grimes
Affiliations: | University of Louisville, Louisville, KY, United States |
Area:
Molecular BiologyGoogle:
"Harry Grimes"
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Publications
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| Muench DE, Grimes HL. (2015) Transcriptional Control of Stem and Progenitor Potential. Current Stem Cell Reports. 1: 139-150 |
| Guo F, Hildeman D, Tripathi P, et al. (2010) Coordination of IL-7 receptor and T-cell receptor signaling by cell-division cycle 42 in T-cell homeostasis. Proceedings of the National Academy of Sciences of the United States of America. 107: 18505-10 |
| Phelan JD, Shroyer NF, Cook T, et al. (2010) Gfi1-cells and circuits: unraveling transcriptional networks of development and disease. Current Opinion in Hematology. 17: 300-7 |
| Montoya-Durango DE, Velu CS, Kazanjian A, et al. (2008) Ajuba functions as a histone deacetylase-dependent co-repressor for autoregulation of the growth factor-independent-1 transcription factor. The Journal of Biological Chemistry. 283: 32056-65 |
| Li-Kroeger D, Witt LM, Grimes HL, et al. (2008) Hox and senseless antagonism functions as a molecular switch to regulate EGF secretion in the Drosophila PNS. Developmental Cell. 15: 298-308 |
| Duan Z, Zarebski A, Montoya-Durango D, et al. (2005) Gfi1 coordinates epigenetic repression of p21Cip/WAF1 by recruitment of histone lysine methyltransferase G9a and histone deacetylase 1. Molecular and Cellular Biology. 25: 10338-51 |
| Park JH, Yu Q, Erman B, et al. (2004) Suppression of IL7Ralpha transcription by IL-7 and other prosurvival cytokines: a novel mechanism for maximizing IL-7-dependent T cell survival. Immunity. 21: 289-302 |
| Doan LL, Porter SD, Duan Z, et al. (2004) Targeted transcriptional repression of Gfi1 by GFI1 and GFI1B in lymphoid cells. Nucleic Acids Research. 32: 2508-19 |
| McGhee L, Bryan J, Elliott L, et al. (2003) Gfi-1 attaches to the nuclear matrix, associates with ETO (MTG8) and histone deacetylase proteins, and represses transcription using a TSA-sensitive mechanism. Journal of Cellular Biochemistry. 89: 1005-18 |
| Doan LL, Kitay MK, Yu Q, et al. (2003) Growth factor independence-1B expression leads to defects in T cell activation, IL-7 receptor alpha expression, and T cell lineage commitment. Journal of Immunology (Baltimore, Md. : 1950). 170: 2356-66 |