Peter S. Aronson

Affiliations: 
Yale University, New Haven, CT 
Area:
Physiology Biology, Molecular Biology
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"Peter Aronson"
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Publications

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Neumeier LI, Thomson RB, Reichel M, et al. (2020) Enteric Oxalate Secretion Mediated by Slc26a6 Defends against Hyperoxalemia in Murine Models of Chronic Kidney Disease. Journal of the American Society of Nephrology : Jasn
Knauf F, Velazquez H, Pfann V, et al. (2018) Characterization of renal NaCl and oxalate transport in Slc26a6-/- mice. American Journal of Physiology. Renal Physiology
Ermer T, Kopp C, Asplin JR, et al. (2017) Impact of Regular or Extended Hemodialysis and Hemodialfiltration on Plasma Oxalate Concentrations in Patients With End-Stage Renal Disease. Kidney International Reports. 2: 1050-1058
Thomson RB, Thomson CL, Aronson PS. (2016) N-glycosylation critically regulates function of oxalate transporter SLC26A6. American Journal of Physiology. Cell Physiology. ajpcell.00171.2016
Knauf F, Thomson RB, Heneghan JF, et al. (2016) Loss of Cystic Fibrosis Transmembrane Regulator Impairs Intestinal Oxalate Secretion. Journal of the American Society of Nephrology : Jasn
Ermer T, Eckardt KU, Aronson PS, et al. (2016) Oxalate, inflammasome, and progression of kidney disease. Current Opinion in Nephrology and Hypertension. 25: 363-71
Mulay SR, Eberhard JN, Pfann V, et al. (2016) Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice. American Journal of Physiology. Renal Physiology. ajprenal.00488.2015
Chen T, Kocinsky HS, Cha B, et al. (2015) Cyclic GMP kinase II (cGKII) inhibits NHE3 by altering its trafficking and phosphorylating NHE3 at three required sites: identification of a multifunctional phosphorylation site. The Journal of Biological Chemistry. 290: 1952-65
Jacques T, Picard N, Miller RL, et al. (2013) Overexpression of pendrin in intercalated cells produces chloride-sensitive hypertension. Journal of the American Society of Nephrology : Jasn. 24: 1104-13
Knauf F, Asplin JR, Granja I, et al. (2013) NALP3-mediated inflammation is a principal cause of progressive renal failure in oxalate nephropathy. Kidney International. 84: 895-901
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