Zamaneh Kassiri, Ph.D.
Affiliations: | 2002 | University of Toronto, Toronto, ON, Canada |
Area:
Animal Physiology Biology, Cell BiologyGoogle:
"Zamaneh Kassiri"Parents
Sign in to add mentorPeter H. Backx | grad student | 2002 | University of Toronto | |
(Reduction of transient outward potassium current and hypertrophy in neonatal rat ventricular myocytes: Role of calcium-dependent signaling pathways.) |
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Publications
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Aujla PK, Hu M, Hartley B, et al. (2022) Loss of ADAM15 Exacerbates Transition to Decompensated Myocardial Hypertrophy and Dilation Through Activation of the Calcineurin Pathway. Hypertension (Dallas, Tex. : 1979) |
Nagalingam RS, Chattopadhyaya S, Al-Hattab DS, et al. (2022) Scleraxis and fibrosis in the pressure-overloaded heart. European Heart Journal |
Chute M, Aujla PK, Li Y, et al. (2022) ADAM15 is required for optimal collagen cross-linking and scar formation following myocardial infarction. Matrix Biology : Journal of the International Society For Matrix Biology |
Fan D, Kassiri Z. (2021) Modulation of Cardiac Fibrosis in and Beyond Cells. Frontiers in Molecular Biosciences. 8: 750626 |
Jana S, Aujla P, Hu M, et al. (2021) Gelsolin is an important mediator of Angiotensin II-induced activation of cardiac fibroblasts and fibrosis. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 35: e21932 |
Oh Y, Yang S, Liu X, et al. (2020) Transcriptomic Bioinformatic Analyses of Atria Uncover Involvement of Pathways Related to Strain and Post-translational Modification of Collagen in Increased Atrial Fibrillation Vulnerability in Intensely Exercised Mice. Frontiers in Physiology. 11: 605671 |
Patel VB, Zhabyeyev P, Chen X, et al. (2018) PI3Kα-regulated gelsolin activity is a critical determinant of cardiac cytoskeletal remodeling and heart disease. Nature Communications. 9: 5390 |
Chen X, Zhabyeyev P, Azad AK, et al. (2018) Endothelial and Cardiomyocyte PI3Kβ Divergently Regulate Cardiac Remodeling in Response to Ischemic Injury. Cardiovascular Research |
Wang W, Patel VB, Parajuli N, et al. (2014) Heterozygote loss of ACE2 is sufficient to increase the susceptibility to heart disease. Journal of Molecular Medicine (Berlin, Germany). 92: 847-58 |
Fan D, Takawale A, Basu R, et al. (2014) Differential role of TIMP2 and TIMP3 in cardiac hypertrophy, fibrosis, and diastolic dysfunction. Cardiovascular Research. 103: 268-80 |