Alicia N. Lyle, Ph.D.
Affiliations: | 2009 | Emory University, Atlanta, GA |
Area:
Cell Biology, Molecular BiologyGoogle:
"Alicia Lyle"Parents
Sign in to add mentor| Kathy K. Griendling | grad student | 2009 | Emory | |
| (Poldip2, a novel regulator of Nox4 and cytoskeletal integrity in vascular smooth muscle cells.) | ||||
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Publications
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| Lyle AN, Taylor WR. (2019) The pathophysiological basis of vascular disease. Laboratory Investigation; a Journal of Technical Methods and Pathology |
| Lok ZSY, Lyle AN. (2019) Osteopontin in Vascular Disease. Arteriosclerosis, Thrombosis, and Vascular Biology. ATVBAHA118311577 |
| Lee GS, Salazar HF, Joseph G, et al. (2018) Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival. Laboratory Investigation; a Journal of Technical Methods and Pathology |
| Okwan-Duodu D, Hansen L, Joseph G, et al. (2018) Impaired Collateral Vessel Formation in Sickle Cell Disease. Arteriosclerosis, Thrombosis, and Vascular Biology |
| Caesar C, Lyle AN, Joseph G, et al. (2017) Cyclic Strain and Hypertension Increase Osteopontin Expression in the Aorta. Cellular and Molecular Bioengineering. 10: 144-152 |
| Amanso A, Lyle AN, Griendling KK. (2017) NADPH Oxidases and Measurement of Reactive Oxygen Species. Methods in Molecular Biology (Clifton, N.J.). 1527: 219-232 |
| Di Marco E, Gray SP, Kennedy K, et al. (2016) NOX4-derived reactive oxygen species limit fibrosis and inhibit proliferation of vascular smooth muscle cells in diabetic atherosclerosis. Free Radical Biology & Medicine |
| Kleinhenz JM, Murphy TC, Pokutta-Paskaleva AP, et al. (2015) Smooth Muscle-Targeted Overexpression of Peroxisome Proliferator Activated Receptor-γ Disrupts Vascular Wall Structure and Function. Plos One. 10: e0139756 |
| Akahori H, Karmali V, Polavarapu R, et al. (2015) CD163 interacts with TWEAK to regulate tissue regeneration after ischaemic injury. Nature Communications. 6: 7792 |
| Remus EW, Sayeed I, Won S, et al. (2015) Progesterone protects endothelial cells after cerebrovascular occlusion by decreasing MCP-1- and CXCL1-mediated macrophage infiltration. Experimental Neurology. 271: 401-408 |