Stephanie A. Parsons, Ph.D.
Affiliations: | 2004 | University of Cincinnati, Cincinnati, OH |
Area:
Molecular BiologyGoogle:
"Stephanie Parsons"Parents
Sign in to add mentor| Jeffery D. Molkentin | grad student | 2004 | University of Cincinnati | |
| (The role of calcineurin in skeletal muscle hypertrophy and fiber type diversity.) | ||||
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| Patel S, Alvarez-Guaita A, Melvin A, et al. (2019) GDF15 Provides an Endocrine Signal of Nutritional Stress in Mice and Humans. Cell Metabolism |
| Wagner KR, Fleckenstein JL, Amato AA, et al. (2008) A phase I/IItrial of MYO-029 in adult subjects with muscular dystrophy. Annals of Neurology. 63: 561-71 |
| Parsons SA, Millay DP, Sargent MA, et al. (2007) Genetic disruption of calcineurin improves skeletal muscle pathology and cardiac disease in a mouse model of limb-girdle muscular dystrophy. The Journal of Biological Chemistry. 282: 10068-78 |
| Parsons SA, Millay DP, Sargent MA, et al. (2006) Age-dependent effect of myostatin blockade on disease severity in a murine model of limb-girdle muscular dystrophy. The American Journal of Pathology. 168: 1975-85 |
| Parsons SA, Millay DP, Wilkins BJ, et al. (2004) Genetic loss of calcineurin blocks mechanical overload-induced skeletal muscle fiber type switching but not hypertrophy. The Journal of Biological Chemistry. 279: 26192-200 |
| Wilkins BJ, Dai YS, Bueno OF, et al. (2004) Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy. Circulation Research. 94: 110-8 |
| Parsons SA, Wilkins BJ, Bueno OF, et al. (2003) Altered skeletal muscle phenotypes in calcineurin Aalpha and Abeta gene-targeted mice. Molecular and Cellular Biology. 23: 4331-43 |
| Braz JC, Bueno OF, Liang Q, et al. (2003) Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NFAT signaling. The Journal of Clinical Investigation. 111: 1475-86 |