Tovah A. Day, Ph.D.
Affiliations: | 2010 | Boston University, Boston, MA, United States |
Area:
Molecular BiologyGoogle:
"Tovah Day"Parents
Sign in to add mentorCyrus Vaziri | grad student | 2010 | Boston University | |
(CDC7-mediated phosphorylation of RAD18 facilitates recruitment of DNA polymerase eta to stalled replication forks.) |
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Publications
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Cong K, MacGilvary N, Lee S, et al. (2024) FANCJ promotes PARP1 activity during DNA replication that is essential in BRCA1 deficient cells. Biorxiv : the Preprint Server For Biology |
Shalwitz R, Day T, Kotsakis Ruehlmann A, et al. (2023) Treatment of Sulfur Mustard Corneal Injury by Augmenting the DNA Damage Response (DDR): A Novel Approach. The Journal of Pharmacology and Experimental Therapeutics |
Durando ML, Menghani SV, Baumann JL, et al. (2022) Four-Year Disease-Free Remission in a Patient With POLE Mutation-Associated Colorectal Cancer Treated Using Anti-PD-1 Therapy. Journal of the National Comprehensive Cancer Network : Jnccn. 20: 218-223 |
Vaziri C, Rogozin IB, Gu Q, et al. (2021) Unravelling roles of error-prone DNA polymerases in shaping cancer genomes. Oncogene |
Layer JV, Debaize L, Van Scoyk A, et al. (2020) Polymerase δ promotes chromosomal rearrangements and imprecise double-strand break repair. Proceedings of the National Academy of Sciences of the United States of America |
Qi H, Price BD, Day TA. (2019) Multiple Roles for Mono- and Poly(ADP-Ribose) in Regulating Stress Responses. Trends in Genetics : Tig. 35: 159-172 |
Layer JV, Cleary JP, Brown AJ, et al. (2018) Parp3 promotes long-range end joining in murine cells. Proceedings of the National Academy of Sciences of the United States of America |
Day TA, Layer JV, Cleary JP, et al. (2017) Corrigendum: PARP3 is a promoter of chromosomal rearrangements and limits G4 DNA. Nature Communications. 8: 15918 |
Day TA, Layer JV, Cleary JP, et al. (2017) PARP3 is a promoter of chromosomal rearrangements and limits G4 DNA. Nature Communications. 8: 15110 |
Choi YE, Meghani K, Brault ME, et al. (2016) Platinum and PARP Inhibitor Resistance Due to Overexpression of MicroRNA-622 in BRCA1-Mutant Ovarian Cancer. Cell Reports |