Yogesh K. Chutake, Ph.D.
Affiliations: | 2011 | University of Oklahoma Health Sciences Center, Oklahoma City, OK, United States |
Area:
Molecular BiologyGoogle:
"Yogesh Chutake"Parents
Sign in to add mentorSanjay Bidichandani | grad student | 2011 | University of Oklahoma Health Sciences Center | |
(RNA-mediated transcriptional gene silencing in Friedreich ataxia.) |
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Publications
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Rodden LN, Chutake YK, Gilliam K, et al. (2021) Methylated and unmethylated epialleles support variegated epigenetic silencing in Friedreich ataxia. Human Molecular Genetics |
Chutake YK, Lam CC, Costello WN, et al. (2016) Reversal of epigenetic promoter silencing in Friedreich ataxia by a class I histone deacetylase inhibitor. Nucleic Acids Research |
Chutake YK, Costello WN, Lam CC, et al. (2015) FXN Promoter Silencing in the Humanized Mouse Model of Friedreich Ataxia. Plos One. 10: e0138437 |
Anjomani Virmouni S, Ezzatizadeh V, Sandi C, et al. (2015) A novel GAA-repeat-expansion-based mouse model of Friedreich's ataxia. Disease Models & Mechanisms. 8: 225-35 |
Chutake YK, Lam C, Costello WN, et al. (2014) Epigenetic promoter silencing in Friedreich ataxia is dependent on repeat length. Annals of Neurology. 76: 522-8 |
Chutake YK, Costello WN, Lam C, et al. (2014) Altered nucleosome positioning at the transcription start site and deficient transcriptional initiation in Friedreich ataxia. The Journal of Biological Chemistry. 289: 15194-202 |
Bidichandani S, Castro A, Chutake Y. (2011) RNA-mediated transcriptional gene silencing in Friedreich ataxia Current Opinion in Biotechnology. 22: S16 |
De Biase I, Chutake YK, Rindler PM, et al. (2009) Epigenetic silencing in Friedreich ataxia is associated with depletion of CTCF (CCCTC-binding factor) and antisense transcription. Plos One. 4: e7914 |
Pollard LM, Chutake YK, Rindler PM, et al. (2007) Deficiency of RecA-dependent RecFOR and RecBCD pathways causes increased instability of the (GAA*TTC)n sequence when GAA is the lagging strand template. Nucleic Acids Research. 35: 6884-94 |