Yogesh K. Chutake, Ph.D.

Affiliations: 
2011 University of Oklahoma Health Sciences Center, Oklahoma City, OK, United States 
Area:
Molecular Biology
Google:
"Yogesh Chutake"

Parents

Sign in to add mentor
Sanjay Bidichandani grad student 2011 University of Oklahoma Health Sciences Center
 (RNA-mediated transcriptional gene silencing in Friedreich ataxia.)
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Rodden LN, Chutake YK, Gilliam K, et al. (2021) Methylated and unmethylated epialleles support variegated epigenetic silencing in Friedreich ataxia. Human Molecular Genetics
Chutake YK, Lam CC, Costello WN, et al. (2016) Reversal of epigenetic promoter silencing in Friedreich ataxia by a class I histone deacetylase inhibitor. Nucleic Acids Research
Chutake YK, Costello WN, Lam CC, et al. (2015) FXN Promoter Silencing in the Humanized Mouse Model of Friedreich Ataxia. Plos One. 10: e0138437
Anjomani Virmouni S, Ezzatizadeh V, Sandi C, et al. (2015) A novel GAA-repeat-expansion-based mouse model of Friedreich's ataxia. Disease Models & Mechanisms. 8: 225-35
Chutake YK, Lam C, Costello WN, et al. (2014) Epigenetic promoter silencing in Friedreich ataxia is dependent on repeat length. Annals of Neurology. 76: 522-8
Chutake YK, Costello WN, Lam C, et al. (2014) Altered nucleosome positioning at the transcription start site and deficient transcriptional initiation in Friedreich ataxia. The Journal of Biological Chemistry. 289: 15194-202
Bidichandani S, Castro A, Chutake Y. (2011) RNA-mediated transcriptional gene silencing in Friedreich ataxia Current Opinion in Biotechnology. 22: S16
De Biase I, Chutake YK, Rindler PM, et al. (2009) Epigenetic silencing in Friedreich ataxia is associated with depletion of CTCF (CCCTC-binding factor) and antisense transcription. Plos One. 4: e7914
Pollard LM, Chutake YK, Rindler PM, et al. (2007) Deficiency of RecA-dependent RecFOR and RecBCD pathways causes increased instability of the (GAA*TTC)n sequence when GAA is the lagging strand template. Nucleic Acids Research. 35: 6884-94
See more...