Kapil Chaudhary, Ph.D.

Affiliations: 
2014 Georgia Regents University 
Area:
Cell Biology
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"Kapil Chaudhary"

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Michael P. Madaio grad student 2014 Georgia Regents University
 (Metabolic signaling in glomerulonephritis and the development of a non-invasive imaging biomarker for glomerular diseases.)
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Publications

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Chaudhary K, Chilakala A, Ananth S, et al. (2019) Renal Iron Accelerates the Progression of Diabetic Nephropathy in HFE (High Fe-iron) Gene Knockout Mouse Model of Iron Overload. American Journal of Physiology. Renal Physiology
Shinde R, Hezaveh K, Halaby MJ, et al. (2018) Apoptotic cell-induced AhR activity is required for immunological tolerance and suppression of systemic lupus erythematosus in mice and humans. Nature Immunology
Chaudhary K, Madaio MP. (2016) Amino acid limitation stress response in inflammation Translational Cancer Research. 5: 220-222
Ravishankar B, Liu H, Shinde R, et al. (2015) The amino acid sensor GCN2 inhibits inflammatory responses to apoptotic cells promoting tolerance and suppressing systemic autoimmunity. Proceedings of the National Academy of Sciences of the United States of America
Shinde R, Shimoda M, Chaudhary K, et al. (2015) B Cell-Intrinsic IDO1 Regulates Humoral Immunity to T Cell-Independent Antigens. Journal of Immunology (Baltimore, Md. : 1950)
Chaudhary K, Shinde R, Liu H, et al. (2015) Amino Acid Metabolism Inhibits Antibody-Driven Kidney Injury by Inducing Autophagy. Journal of Immunology (Baltimore, Md. : 1950). 194: 5713-24
Ravishankar B, Shinde R, Liu H, et al. (2014) Marginal zone CD169+ macrophages coordinate apoptotic cell-driven cellular recruitment and tolerance. Proceedings of the National Academy of Sciences of the United States of America. 111: 4215-20
Chaudhary K, Kleven DT, McGaha TL, et al. (2013) A human monoclonal antibody against the collagen type IV α3NC1 domain is a non-invasive optical biomarker for glomerular diseases. Kidney International. 84: 403-8
Kvirkvelia N, McMenamin M, Chaudhary K, et al. (2013) Prostaglandin E2 promotes cellular recovery from established nephrotoxic serum nephritis in mice, prosurvival, and regenerative effects on glomerular cells. American Journal of Physiology. Renal Physiology. 304: F463-70
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