Bryant Boulianne, Ph.D.
Affiliations: | 2013 | Immunology | University of Toronto, Toronto, ON, Canada |
Area:
Immunology, Cell BiologyGoogle:
"Bryant Boulianne"Parents
Sign in to add mentor| Jennifer Gommerman | grad student | 2013 | University of Toronto | |
| (Investigating the Roles of Follicular Dendritic Cells and Activation-Induced Deaminase in Germinal Centers.) | ||||
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Publications
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| Pfeifer M, Brem R, Lippert TP, et al. (2019) SSB1/SSB2 Proteins Safeguard B Cell Development by Protecting the Genomes of B Cell Precursors. Journal of Immunology (Baltimore, Md. : 1950) |
| Boulianne B, Robinson ME, May PC, et al. (2017) Lineage-Specific Genes Are Prominent DNA Damage Hotspots during Leukemic Transformation of B Cell Precursors. Cell Reports. 18: 1687-1698 |
| Paradis M, Mindt BC, Duerr CU, et al. (2014) A TNF-α-CCL20-CCR6 axis regulates Nod1-induced B cell responses. Journal of Immunology (Baltimore, Md. : 1950). 192: 2787-99 |
| Boulianne B, Rojas OL, Haddad D, et al. (2013) AID and caspase 8 shape the germinal center response through apoptosis. Journal of Immunology (Baltimore, Md. : 1950). 191: 5840-7 |
| Boulianne B, Le MX, Ward LA, et al. (2013) AID-expressing germinal center B cells cluster normally within lymph node follicles in the absence of FDC-M1+ CD35+ follicular dendritic cells but dissipate prematurely. Journal of Immunology (Baltimore, Md. : 1950). 191: 4521-30 |
| Galicia G, Boulianne B, Pikor N, et al. (2013) Secondary B cell receptor diversification is necessary for T cell mediated neuro-inflammation during experimental autoimmune encephalomyelitis. Plos One. 8: e61478 |
| Boulianne B, Porfilio EA, Pikor N, et al. (2012) Lymphotoxin-sensitive microenvironments in homeostasis and inflammation. Frontiers in Immunology. 3: 243 |
| Green B, Belcheva A, Nepal RM, et al. (2011) The mismatch repair pathway functions normally at a non-AID target in germinal center B cells. Blood. 118: 3013-8 |
| Zaheen A, Boulianne B, Parsa JY, et al. (2009) AID constrains germinal center size by rendering B cells susceptible to apoptosis. Blood. 114: 547-54 |