Neta Shlezinger
Affiliations: | 2014-2019 | Hebrew University, Jerusalem, Jerusalem, Israel |
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"Neta Shlezinger"
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| Aufiero MA, Shlezinger N, Gjonbalaj M, et al. (2023) Dectin-1/CARD9 induction of the TFEB and TFE3 gene network is dispensable for phagocyte anti- activity in the lung. Infection and Immunity. e0021723 |
| Yehuda A, Malach E, Vanunu Ofri S, et al. (2023) The quorum-sensing peptidic inhibitor rescues host immune system eradication: A novel infectivity mechanism. Proceedings of the National Academy of Sciences of the United States of America. 120: e2301045120 |
| Aufiero MA, Shlezinger N, Gjonbalaj M, et al. (2023) Dectin-1/CARD9-induction of the TFEB and TFE3 gene network is dispensable for phagocyte anti- activity in the lung. Biorxiv : the Preprint Server For Biology |
| Shlezinger N, Hohl TM. (2021) Mitochondrial Reactive Oxygen Species Enhance Alveolar Macrophage Activity against Aspergillus fumigatus but Are Dispensable for Host Protection. Msphere. e0026021 |
| Shlezinger N, Fites JS, Klein BS, et al. (2021) Fungal Bioreporters to Monitor Outcomes of Aspergillus: Host-Cell Interactions. Methods in Molecular Biology (Clifton, N.J.). 2260: 121-132 |
| Fites JS, Shlezinger N, Hohl TM, et al. (2021) Fungal Bioreporters to Monitor Outcomes of Blastomyces: Host-Cell Interactions. Methods in Molecular Biology (Clifton, N.J.). 2260: 111-119 |
| Shlezinger N, Irmer H, Dhingra S, et al. (2018) Response to Comment on "Sterilizing immunity in the lung relies on targeting fungal apoptosis-like programmed cell death". Science (New York, N.Y.). 360 |
| Shlezinger N, Irmer H, Dhingra S, et al. (2017) Sterilizing immunity in the lung relies on targeting fungal apoptosis-like programmed cell death. Science (New York, N.Y.). 357: 1037-1041 |
| Sharon A, Shlezinger N. (2013) Fungi infecting plants and animals: killers, non-killers, and cell death. Plos Pathogens. 9: e1003517 |
| Shlezinger N, Doron A, Sharon A. (2011) Apoptosis-like programmed cell death in the grey mould fungus Botrytis cinerea: genes and their role in pathogenicity. Biochemical Society Transactions. 39: 1493-8 |