Kelly A. Fader

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2016- Michigan State University, East Lansing, MI 
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"Kelly Fader"
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Cholico GN, Orlowska K, Fling RR, et al. (2023) Consequences of reprogramming acetyl-CoA metabolism by 2,3,7,8-tetrachlorodibenzo-p-dioxin in the mouse liver. Scientific Reports. 13: 4138
Cholico GN, Fling RR, Zacharewski NA, et al. (2021) Thioesterase induction by 2,3,7,8-tetrachlorodibenzo-p-dioxin results in a futile cycle that inhibits hepatic β-oxidation. Scientific Reports. 11: 15689
Fling RR, Doskey CM, Fader KA, et al. (2020) 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) dysregulates hepatic one carbon metabolism during the progression of steatosis to steatohepatitis with fibrosis in mice. Scientific Reports. 10: 14831
Nault R, Fader KA, Bhattacharya S, et al. (2020) Single nuclei RNA sequencing assessment of the hepatic effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin. Cellular and Molecular Gastroenterology and Hepatology
Doskey CM, Fader KA, Nault R, et al. (2020) 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) alters hepatic polyunsaturated fatty acid metabolism and eicosanoid biosynthesis in female Sprague-Dawley rats. Toxicology and Applied Pharmacology. 115034
Dornbos P, Jurgelewicz A, Fader KA, et al. (2019) Characterizing the Role of HMG-CoA Reductase in Aryl Hydrocarbon Receptor-Mediated Liver Injury in C57BL/6 Mice. Scientific Reports. 9: 15828
Fader KA, Nault R, Doskey CM, et al. (2019) 2,3,7,8-Tetrachlorodibenzo-p-dioxin abolishes circadian regulation of hepatic metabolic activity in mice. Scientific Reports. 9: 6514
Nault R, Doskey CM, Fader KA, et al. (2018) Comparison of hepatic NRF2 and AHR binding in 2,3,7,8-tetrachlorodibenzo--dioxin (TCDD) treated mice demonstrates NRF2-independent PKM2 induction. Molecular Pharmacology
Fader KA, Nault R, Raehtz S, et al. (2018) 2,3,7,8-Tetrachlorodibenzo-p-dioxin dose-dependently increases bone mass and decreases marrow adiposity in juvenile mice. Toxicology and Applied Pharmacology
Fader KA, Nault R, Kirby MP, et al. (2018) Corrigendum to "Convergence of hepcidin deficiency, systemic iron overloading, heme accumulation, and REV-ERBα/β activation in aryl hydrocarbon receptor-elicited hepatotoxicity" [Toxicol Appl Pharmacol. 321 (2017) 1-17]. Toxicology and Applied Pharmacology
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