Meng Wang, Ph.D.

Affiliations: 
Baylor College of Medicine, Houston, TX 
Website:
http://mengwanglab.org/
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"Meng Wang"

Children

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Marzia Savini grad student Baylor College of Medicine
Tao Chen post-doc 2019-2023 (Biomechanics Tree)
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Publications

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Fu H, Jin J, Xiao S, et al. (2025) The analysis of distinctive lipid metabolism characteristics in dry-cured beef during processing. Food Chemistry: X. 28: 102540
Chen T, Savini M, Wang MC. (2025) Unlocking in vivo metabolic insights with vibrational microscopy. Nature Methods. 22: 886-889
Deng R, Zhu Y, Liu K, et al. (2024) Genetic loss of Nrf1 and Nrf2 leads to distinct metabolism reprogramming of HepG2 cells by opposing regulation of the PI3K-AKT-mTOR signalling pathway. Bioorganic Chemistry. 145: 107212
Deng R, Zheng Z, Hu S, et al. (2023) Loss of Nrf1 rather than Nrf2 leads to inflammatory accumulation of lipids and reactive oxygen species in human hepatoma cells, which is alleviated by 2-bromopalmitate. Biochimica Et Biophysica Acta. Molecular Cell Research. 119644
Lee YT, Savini M, Chen T, et al. (2023) Mitochondrial GTP metabolism controls reproductive aging in C. elegans. Developmental Cell
Papsdorf K, Miklas JW, Hosseini A, et al. (2023) Author Correction: Lipid droplets and peroxisomes are co-regulated to drive lifespan extension in response to mono-unsaturated fatty acids. Nature Cell Biology
Gedam M, Comerota MM, Propson NE, et al. (2023) Complement C3aR depletion reverses HIF-1α-induced metabolic impairment and enhances microglial response to Aβ pathology. The Journal of Clinical Investigation. 133
Papsdorf K, Miklas JW, Hosseini A, et al. (2023) Lipid droplets and peroxisomes are co-regulated to drive lifespan extension in response to mono-unsaturated fatty acids. Nature Cell Biology
Lee YT, Savini M, Chen T, et al. (2023) Mitochondrial GTP Metabolism Regulates Reproductive Aging. Biorxiv : the Preprint Server For Biology
Comerota M, Gedam M, Xiong W, et al. (2023) Oleoylethanolamide facilitates PPARa and TFEB signaling and attenuates Ab pathology in a mouse model of Alzheimer's disease. Research Square
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