Tom Curran
Affiliations: | Children's Mercy Kansas City | ||
| 1984-1995 | Roche Institute of Molecular Biology |
Area:
Biology, Cancer Research, NeuroscienceWebsite:
https://bionexuskc.org/staff/tom-curran-phd-frs/Google:
"Tom Curran"Bio:
https://doi.org/10.4161/cbt.2.4.449
https://scholar.google.com/citations?user=cgHlvUQAAAAJ&hl=en
Children
Sign in to add trainee| Tom Klaus Kerppola | post-doc | Roche Institute of Molecular Biology (Chemistry Tree) |
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Publications
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| Huang Y, Clarke F, Karimi M, et al. (2015) CRK proteins selectively regulate T cell migration into inflamed tissues. The Journal of Clinical Investigation. 125: 1019-32 |
| Gurung B, Feng Z, Iwamoto DV, et al. (2013) Menin epigenetically represses Hedgehog signaling in MEN1 tumor syndrome. Cancer Research. 73: 2650-8 |
| Hallock PT, Xu CF, Park TJ, et al. (2010) Dok-7 regulates neuromuscular synapse formation by recruiting Crk and Crk-L. Genes & Development. 24: 2451-61 |
| Park TJ, Curran T. (2008) Crk and Crk-like play essential overlapping roles downstream of disabled-1 in the Reelin pathway. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 13551-62 |
| Lee Y, Kawagoe R, Sasai K, et al. (2007) Loss of suppressor-of-fused function promotes tumorigenesis. Oncogene. 26: 6442-7 |
| Park TJ, Boyd K, Curran T. (2006) Cardiovascular and craniofacial defects in Crk-null mice. Molecular and Cellular Biology. 26: 6272-82 |
| Liu J, Wang J, Huang Q, et al. (2006) Gene expression profiles of mouse retinas during the second and third postnatal weeks. Brain Research. 1098: 113-25 |
| Magdaleno S, Jensen P, Brumwell CL, et al. (2006) BGEM: an in situ hybridization database of gene expression in the embryonic and adult mouse nervous system. Plos Biology. 4: e86 |
| Uziel T, Zindy F, Xie S, et al. (2005) The tumor suppressors Ink4c and p53 collaborate independently with Patched to suppress medulloblastoma formation. Genes & Development. 19: 2656-67 |
| Ordway JM, Fenster SD, Ruan H, et al. (2005) A transcriptome map of cellular transformation by the fos oncogene. Molecular Cancer. 4: 19 |