Alf Herzig
Affiliations: | Max Planck Institute for Biophysical Chemistry, Göttingen, Niedersachsen, Germany |
Area:
ChromatinGoogle:
"Alf Herzig"Mean distance: 8.78 (cluster 10) | S | N | B | C | P |
Cross-listing: FlyTree
Parents
Sign in to add mentor| Christian F. Lehner | research assistant | 1994-1996 | Freidrich Miescher Institute (FlyTree) |
| Christian F. Lehner | grad student | 1999-2003 | (FlyTree) |
| Herbert Jäckle | post-doc | 2003- | Max Planck Institute for Biophysical Chemistry (FlyTree) |
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Publications
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| Sollberger G, Choidas A, Burn GL, et al. (2018) Gasdermin D plays a vital role in the generation of neutrophil extracellular traps. Science Immunology. 3 |
| Schnorrenberg S, Grotjohann T, Vorbrüggen G, et al. (2016) In vivo super-resolution RESOLFT microscopy of Drosophila melanogaster. Elife. 5 |
| Günesdogan U, Jäckle H, Herzig A. (2014) Histone supply regulates S phase timing and cell cycle progression. Elife. 3: e02443 |
| Yakulov T, Günesdogan U, Jäckle H, et al. (2014) Bällchen participates in proliferation control and prevents the differentiation of Drosophila melanogaster neuronal stem cells. Biology Open. 3: 881-6 |
| Herzig B, Yakulov TA, Klinge K, et al. (2014) Bällchen is required for self-renewal of germline stem cells in Drosophila melanogaster. Biology Open. 3: 510-21 |
| Günesdogan U, Jäckle H, Herzig A. (2014) Author response: Histone supply regulates S phase timing and cell cycle progression Elife |
| Pengelly AR, Copur Ö, Jäckle H, et al. (2013) A histone mutant reproduces the phenotype caused by loss of histone-modifying factor Polycomb. Science (New York, N.Y.). 339: 698-9 |
| Gajula Balija MB, Griesinger C, Herzig A, et al. (2011) Pre-fibrillar α-synuclein mutants cause Parkinson's disease-like non-motor symptoms in Drosophila. Plos One. 6: e24701 |
| Günesdogan U, Jäckle H, Herzig A. (2010) A genetic system to assess in vivo the functions of histones and histone modifications in higher eukaryotes. Embo Reports. 11: 772-6 |
| Karpinar DP, Balija MB, Kügler S, et al. (2009) Pre-fibrillar alpha-synuclein variants with impaired beta-structure increase neurotoxicity in Parkinson's disease models. The Embo Journal. 28: 3256-68 |