Michael John Palladino
Affiliations: | Pharmacology | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
NeurodegenerationWebsite:
http://www.pharmacology.us/Faculty/MichaelPalladinoGoogle:
"Michael John Palladino"Mean distance: 15.35 (cluster 11) | S | N | B | C | P |
Cross-listing: Neurotree
Parents
Sign in to add mentor| Robert A.G. Reenan | grad student | 2000 | University of Connecticut (FlyTree) | |
| (dADAR, a Drosophila RNA editing enzyme, modifies ion channel transcripts and is required for normal behavior and nervous system integrity.) | ||||
| Barry Ganetzky | post-doc | 2003 | UW Madison (Neurotree) | |
Children
Sign in to add trainee| Adam Charles Frank | research assistant | 2003-2007 | Pitt (Neurotree) |
| Bartholomew P Roland | grad student | 2008-2014 | University of Pittsburgh School of Medicine |
BETA: Related publications
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Publications
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| Yang L, Borne F, Betz A, et al. (2023) Predatory fireflies and their toxic firefly prey have evolved distinct toxin resistance strategies. Current Biology : Cb |
| Taverner AM, Yang L, Barile ZJ, et al. (2019) Adaptive substitutions underlying cardiac glycoside insensitivity in insects exhibit epistasis . Elife. 8 |
| Roland BP, Richards KR, Hrizo SL, et al. (2019) Missense variant in TPI1 (Arg189Gln) causes neurologic deficits through structural changes in the triosephosphate isomerase catalytic site and reduced enzyme levels in vivo. Biochimica Et Biophysica Acta. Molecular Basis of Disease |
| Fogle KJ, Mobini CL, Paseos AS, et al. (2019) Sleep and circadian defects in a model of mitochondrial encephalomyopathy. Neurobiology of Sleep and Circadian Rhythms. 6: 44-52 |
| Fogle KJ, Smith AR, Satterfield SL, et al. (2019) Ketogenic and anaplerotic dietary modifications ameliorate seizure activity in Drosophila models of mitochondrial encephalomyopathy and glycolytic enzymopathy. Molecular Genetics and Metabolism |
| Taverner AM, Yang L, Barile ZJ, et al. (2019) Author response: Adaptive substitutions underlying cardiac glycoside insensitivity in insects exhibit epistasis in vivo Elife |
| Drombosky KW, Rode S, Kodali R, et al. (2018) Mutational analysis implicates the amyloid fibril as the toxic entity in Huntington's disease. Neurobiology of Disease |
| Markantone DM, Towheed A, Crain AT, et al. (2018) Protein coding mitochondrial-targeted RNAs rescue mitochondrial disease in vivo. Neurobiology of Disease |
| Fogle KJ, Hertzler JI, Shon JH, et al. (2016) The ATP-sensitive K channel is seizure protective and required for effective dietary therapy in a model of mitochondrial encephalomyopathy. Journal of Neurogenetics. 1-12 |
| Roland BP, Zeccola AM, Larsen SB, et al. (2016) Structural and Genetic Studies Demonstrate Neurologic Dysfunction in Triosephosphate Isomerase Deficiency Is Associated with Impaired Synaptic Vesicle Dynamics. Plos Genetics. 12: e1005941 |