Ross W. Milne
Affiliations: | University of Ottawa, Ottawa, ON, Canada |
Area:
BiochemistryWebsite:
http://www.med.uottawa.ca/patho/eng/milne.htmlGoogle:
"Ross William Milne"Bio:
https://www.ottawaheart.ca/physician-researcher-profile/milne-ross
https://www.ottawaheart.ca/sites/default/files/files/csv/ross-milne-cv.pdf
https://books.google.com/books?id=xDDgAAAAMAAJ
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Publications
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| Rafatian N, Milne RW, Leenen FH, et al. (2013) Role of renin-angiotensin system in activation of macrophages by modified lipoproteins. American Journal of Physiology. Heart and Circulatory Physiology. 305: H1309-20 |
| Rafatian N, Karunakaran D, Rayner KJ, et al. (2013) Cathepsin G deficiency decreases complexity of atherosclerotic lesions in apolipoprotein E-deficient mice. American Journal of Physiology. Heart and Circulatory Physiology. 305: H1141-8 |
| Caravaggio JW, Hasu M, MacLaren R, et al. (2013) Insulin-degrading enzyme deficiency in bone marrow cells increases atherosclerosis in LDL receptor-deficient mice. Cardiovascular Pathology : the Official Journal of the Society For Cardiovascular Pathology. 22: 458-64 |
| Sneck M, Nguyen SD, Pihlajamaa T, et al. (2012) Conformational changes of apoB-100 in SMase-modified LDL mediate formation of large aggregates at acidic pH. Journal of Lipid Research. 53: 1832-9 |
| Bancells C, Benítez S, Ordóñez-Llanos J, et al. (2011) Immunochemical analysis of the electronegative LDL subfraction shows that abnormal N-terminal apolipoprotein B conformation is involved in increased binding to proteoglycans. The Journal of Biological Chemistry. 286: 1125-33 |
| Liao W, Strube RW, Milne RW, et al. (2008) Cloning of apoB intrabodies: specific knockdown of apoB in HepG2 cells. Biochemical and Biophysical Research Communications. 373: 235-40 |
| Kiss RS, Kavaslar N, Okuhira K, et al. (2007) Genetic etiology of isolated low HDL syndrome: incidence and heterogeneity of efflux defects. Arteriosclerosis, Thrombosis, and Vascular Biology. 27: 1139-45 |
| Heath RB, Karpe F, Milne RW, et al. (2007) Dietary fatty acids make a rapid and substantial contribution to VLDL-triacylglycerol in the fed state. American Journal of Physiology. Endocrinology and Metabolism. 292: E732-9 |
| Hodson L, Fielding B, Milne R, et al. (2007) WO9-OR-5 INSULIN RESISTANCE REDUCES THE CONTRIBUTION OF SYSTEMIC PLASMA NON-ESTERIFIED FATTY ACIDS INTO VERY LOW-DENSITY LIPOPROTEIN TRIACYLGLYCEROL Atherosclerosis Supplements. 8: 9-10 |
| Nguyen AT, Hirama T, Chauhan V, et al. (2006) Binding characteristics of a panel of monoclonal antibodies against the ligand binding domain of the human LDLr. Journal of Lipid Research. 47: 1399-405 |